Jacob R Enriquez
Overview
Explore the profile of Jacob R Enriquez including associated specialties, affiliations and a list of published articles.
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Articles
16
Citations
104
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0
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Recent Articles
1.
Tominaga K, Kechele D, Sanchez J, Vales S, Jurickova I, Roman L, et al.
Cell Mol Gastroenterol Hepatol
. 2024 Dec;
19(4):101444.
PMID: 39701210
Background & Aims: Organs of the gastrointestinal tract contain tissue-resident immune cells that function during tissue development, homeostasis, and disease. However, most published human organoid model systems lack resident immune...
2.
Nargis T, Muralidharan C, Enriquez J, Wang J, Kaylan K, Chakraborty A, et al.
JCI Insight
. 2024 Nov;
9(24).
PMID: 39531315
Type 1 diabetes (T1D) is characterized by the autoimmune destruction of insulin-producing β cells and involves an interplay between β cells and cells of the innate and adaptive immune systems....
3.
Sanchez J, Rankin S, Paul E, McCauley H, Kechele D, Enriquez J, et al.
Development
. 2024 Sep;
151(18).
PMID: 39302049
No abstract available.
4.
Nargis T, Muralidharan C, Enriquez J, Wang J, Kaylan K, Chakraborty A, et al.
bioRxiv
. 2024 Aug;
PMID: 39091839
Type 1 diabetes (T1D) is characterized by the autoimmune destruction of insulin-producing β cells and involves an interplay between β cells and cells of the innate and adaptive immune systems....
5.
Muralidharan C, Huang F, Enriquez J, Wang J, Nelson J, Nargis T, et al.
bioRxiv
. 2024 Jun;
PMID: 38895427
Preventing the onset of autoimmune type 1 diabetes (T1D) is feasible through pharmacological interventions that target molecular stress-responsive mechanisms. Cellular stresses, such as nutrient deficiency, viral infection, or unfolded proteins,...
6.
Muralidharan C, Huang F, Enriquez J, Wang J, Nelson J, Nargis T, et al.
J Clin Invest
. 2024 Jun;
134(16).
PMID: 38889047
Preventing the onset of autoimmune type 1 diabetes (T1D) is feasible through pharmacological interventions that target molecular stress-responsive mechanisms. Cellular stresses, such as nutrient deficiency, viral infection, or unfolded proteins,...
7.
Xu J, Harris-Kawano A, Enriquez J, Mirmira R, Sims E
bioRxiv
. 2024 Apr;
PMID: 38659945
Article Highlights: Mechanisms connecting β cell stress to changes in extracellular vesicle (EV) cargo and potential impacts on diabetes are poorly defined. Does β cell inflammatory stress engage neutral sphingomyelinase...
8.
Sanchez J, Rankin S, Paul E, McCauley H, Kechele D, Enriquez J, et al.
Development
. 2024 Apr;
151(9).
PMID: 38587174
The gastrointestinal (GI) tract is complex and consists of multiple organs with unique functions. Rare gene variants can cause congenital malformations of the human GI tract, although the molecular basis...
9.
Munera J, Kechele D, Bouffi C, Qu N, Jing R, Maity P, et al.
Cell Stem Cell
. 2023 Nov;
30(11):1434-1451.e9.
PMID: 37922878
Most organs have tissue-resident immune cells. Human organoids lack these immune cells, which limits their utility in modeling many normal and disease processes. Here, we describe that pluripotent stem cell-derived...
10.
Sims E, Kulkarni A, Hull A, Woerner S, Cabrera S, Mastrandrea L, et al.
Cell Rep Med
. 2023 Nov;
4(11):101261.
PMID: 37918404
In preclinical models, α-difluoromethylornithine (DFMO), an ornithine decarboxylase (ODC) inhibitor, delays the onset of type 1 diabetes (T1D) by reducing β cell stress. However, the mechanism of DFMO action and...