J P Tremblay
Overview
Explore the profile of J P Tremblay including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
147
Citations
1363
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Torrente Y, El Fahime E, Caron N, Bresolin N, Tremblay J
Cell Transplant
. 2017 Aug;
9(4):539-549.
PMID: 28853939
The effect of pretreatments of host muscles with metalloproteinases (MMPs) or with notexin on the migration of transplanted myoblasts was investigated. Transgenic TnILacZ mice in which the β-galactosidase gene is...
2.
Ouellet D, Cherif K, Rousseau J, Tremblay J
Gene Ther
. 2016 Dec;
24(5):265-274.
PMID: 28024081
The Friedreich ataxia is a monogenic disease due to a hyperexpanded GAA triplet located within the first intron of the frataxin gene that causes transcriptional issues. The resulting frataxin protein...
3.
Chapdelaine P, Gerard C, Sanchez N, Cherif K, Rousseau J, Ouellet D, et al.
Gene Ther
. 2016 Apr;
23(7):606-14.
PMID: 27082765
Artificially designed transcription activator-like effector (TALE) proteins fused to a transcription activation domain (TAD), such as VP64, are able to activate specific eukaryotic promoters. They thus provide a good tool...
4.
Chapdelaine P, Pichavant C, Rousseau J, Paques F, Tremblay J
Gene Ther
. 2010 Apr;
17(7):846-58.
PMID: 20393509
Mutations in Duchenne muscular dystrophy (DMD) are either inducing a nonsense codon or a frameshift. Meganucleases (MGNs) can be engineered to induce double-strand breaks (DSBs) at specific DNA sequences. These...
5.
Benabdallah B, Bouchentouf M, Rousseau J, Tremblay J
Cell Transplant
. 2009 Jun;
18(7):709-18.
PMID: 19520047
Duchenne muscular dystrophy is caused by the absence of functional dystrophin, leading to the myofiber membrane instability and progressive muscle atrophy. Myoblast transplantation in dystrophic muscles is a potential therapy,...
6.
Bouchentouf M, Benabdallah B, Bigey P, Yau T, Scherman D, Tremblay J
Gene Ther
. 2007 Dec;
15(6):404-14.
PMID: 18079754
Muscle precursor cell (myoblasts) transplantation is considered as a potential approach to restore dystrophin expression in Duchenne muscular dystrophy (DMD) patients. The study purpose was to verify the implication of...
7.
A synthetic mechano growth factor E Peptide enhances myogenic precursor cell transplantation success
Mills P, Dominique J, Lafreniere J, Bouchentouf M, Tremblay J
Am J Transplant
. 2007 Sep;
7(10):2247-59.
PMID: 17845560
Myogenic precursor cell (MPC) transplantation is a good strategy to introduce dystrophin expression in muscles of Duchenne muscular dystrophy (DMD) patients. Insulin-like growth factor (IGF-1) promotes MPC activities, such as...
8.
Bouchentouf M, Benabdallah B, Rousseau J, Schwartz L, Tremblay J
Am J Transplant
. 2007 May;
7(6):1491-505.
PMID: 17511679
Seventy-five percent of the myoblasts transplanted in the mouse muscle die during the first 4 days following transplantation. The purpose of this study was to determine if anoikis plays a...
9.
Quenneville S, Chapdelaine P, Rousseau J, Tremblay J
Gene Ther
. 2006 Dec;
14(6):514-22.
PMID: 17167499
Duchenne muscular dystrophy (DMD) is the most severe muscular dystrophy. It is caused by the absence of dystrophin in muscle fibers. The autologous transplantation of genetically corrected muscle precursor cells...
10.
Lafreniere J, Mills P, Bouchentouf M, Tremblay J
Exp Cell Res
. 2006 Feb;
312(7):1127-41.
PMID: 16466711
Different molecules are available to recruit new neighboring myogenic cells to the site of regeneration. Formerly called B cell stimulatory factor-1, IL-4 can now be included in the list of...