J M Penninger
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Explore the profile of J M Penninger including associated specialties, affiliations and a list of published articles.
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119
Citations
12366
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Recent Articles
1.
Benzoni P, Arici M, Giannetti F, Cospito A, Prevostini R, Volani C, et al.
Acta Physiol (Oxf)
. 2024 May;
240(8):e14160.
PMID: 38747650
Aim: Striatin (Strn) is a scaffold protein expressed in cardiomyocytes (CMs) and alteration of its expression are described in various cardiac diseases. However, the alteration underlying its pathogenicity have been...
2.
Manent J, Banerjee S, de Matos Simoes R, Zoranovic T, Mitsiades C, Penninger J, et al.
Oncogene
. 2017 Oct;
36(40):5658-5660.
PMID: 28980625
This corrects the article DOI: 10.1038/onc.2017.175.
3.
Manent J, Banerjee S, de Matos Simoes R, Zoranovic T, Mitsiades C, Penninger J, et al.
Oncogene
. 2017 Jun;
36(40):5576-5592.
PMID: 28581519
Activation of Ras signalling occurs in ~30% of human cancers; however, activated Ras alone is not sufficient for tumourigenesis. In a screen for tumour suppressors that cooperate with oncogenic Ras...
4.
Openshaw R, Thomson D, Penninger J, Pratt J, Morris B
Psychopharmacology (Berl)
. 2016 Oct;
234(2):293-305.
PMID: 27774567
Rationale: Members of the c-Jun N-terminal kinase (JNK) family of mitogen-activated protein (MAP) kinases, and the upstream kinase MKK7, have all been strongly linked with synaptic plasticity and with the...
5.
Galluzzi L, Bravo-San Pedro J, Vitale I, Aaronson S, Abrams J, Adam D, et al.
Cell Death Differ
. 2014 Sep;
22(1):58-73.
PMID: 25236395
Cells exposed to extreme physicochemical or mechanical stimuli die in an uncontrollable manner, as a result of their immediate structural breakdown. Such an unavoidable variant of cellular demise is generally...
6.
Offord E, Karagounis L, Vidal K, Fielding R, Meydani S, Penninger J
J Nutr Health Aging
. 2013 Oct;
17(8):712-6.
PMID: 24097029
No abstract available.
7.
Badger-Brown K, Gillis L, Bailey M, Penninger J, Barber D
Leukemia
. 2012 Nov;
27(5):1146-54.
PMID: 23160449
BCR-ABL induces chronic myeloid leukemia (CML) through the aberrant regulation of multiple signaling substrates. Previous research has shown that BCR-ABL mediates down-modulation of CBL-B protein levels. A murine bone marrow...
8.
Wohrle F, Halbach S, Aumann K, Schwemmers S, Braun S, Auberger P, et al.
Leukemia
. 2012 Aug;
27(1):118-29.
PMID: 22858987
Grb2-associated binder 2 (Gab2) serves as a critical amplifier in the signaling network of Bcr-Abl, the driver of chronic myeloid leukemia (CML). Despite the success of tyrosine kinase inhibitors (TKIs)...
9.
Rey-Parra G, Vadivel A, Coltan L, Hall A, Eaton F, Schuster M, et al.
J Mol Med (Berl)
. 2012 Jan;
90(6):637-47.
PMID: 22246130
Despite substantial progress, mortality and morbidity of the acute respiratory distress syndrome (ARDS), a severe form of acute lung injury (ALI), remain unacceptably high. There is no effective treatment for...
10.
Meixner A, Karreth F, Kenner L, Penninger J, Wagner E
Cell Death Differ
. 2010 Mar;
17(9):1409-19.
PMID: 20300111
Jun is essential for fetal development, as fetuses lacking Jun die at mid-gestation with multiple cellular defects in liver and heart. Embryos expressing JunD in place of Jun (Jun(d/d)) can...