J A CORTNER
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Explore the profile of J A CORTNER including associated specialties, affiliations and a list of published articles.
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51
Citations
305
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Recent Articles
1.
Tershakovec A, Jawad A, Stallings V, CORTNER J, Zemel B, Shannon B
J Pediatr
. 1998 Apr;
132(3 Pt 1):414-20.
PMID: 9544893
Objective: To describe the age-related changes in cardiovascular disease risk factors in young, hypercholesterolemic (HC) children. Methods: Hypercholesterolemic (n = 227) and nonhypercholesterolemic (NHC) (n = 80) children between the...
2.
Tershakovec A, Shannon B, Achterberg C, McKenzie J, Martel J, Smiciklas-Wright H, et al.
Am J Public Health
. 1998 Mar;
88(2):258-61.
PMID: 9491017
Objectives: This study evaluated retention of the effect of a home-based, practitioner-initiated nutrition education model. Methods: Children with elevated low-density lipoprotein (LDL) cholesterol levels were randomly assigned to one of...
3.
Dixon L, Shannon B, Tershakovec A, Bennett M, Coates P, CORTNER J
Am J Clin Nutr
. 1997 Nov;
66(5):1207-17.
PMID: 9356540
We examined the effects of family history of coronary artery disease (CAD), apolipoprotein E (apo E) phenotype, and lipoprotein(a) [Lp(a)] on the response of plasma lipids to change in dietary...
4.
Le N, Coates P, Gallagher P, CORTNER J
Metabolism
. 1997 May;
46(5):584-94.
PMID: 9160828
Orally ingested vitamin A (retinol) is incorporated into intestinal chylomicrons (CHYLO) in the form of retinyl esters (RE) along with newly absorbed dietary triglycerides (TG). As the intestinal lipoproteins undergo...
5.
Shamir R, Tershakovec A, Gallagher P, Liacouras C, Hayman L, CORTNER J
Atherosclerosis
. 1996 Mar;
121(1):85-91.
PMID: 8678927
Background: Familial combined hyperlipidemia (FCHL) has been described as the leading cause of familial hyperlipidemia. FCHL is dominantly inherited, occurs in at least 1% of the population, and is responsible...
6.
Ameis D, Brockmann G, KNOBLICH R, Merkel M, Ostlund Jr R, Yang J, et al.
J Lipid Res
. 1995 Feb;
36(2):241-50.
PMID: 7751811
Cholesteryl ester storage disease (CESD) results from inherited deficiencies of the lysosomal hydrolase, acid lipase (LAL; E.C. 3.1.1.13). To establish the molecular defects in LAL deficiency, two unrelated probands with...
7.
Shannon B, Tershakovec A, Martel J, Achterberg C, CORTNER J, Stallings V, et al.
Pediatrics
. 1994 Dec;
94(6 Pt 1):923-7.
PMID: 7971012
Objective: To assess the effects of a home-based, parent-child autotutorial (PCAT) dietary education program on the dietary knowledge, lipid consumption, and plasma low density lipoprotein-cholesterol (LDL-C) of 4- to 10-year-old...
8.
Familial combined hyperlipidemia in children: clinical expression, metabolic defects, and management
CORTNER J, Coates P, Liacouras C, Jarvik G
Curr Probl Pediatr
. 1994 Oct;
24(9):295-305.
PMID: 7859485
The first evidence that elevation of plasma levels of cholesterol is a risk factor for the development of atherosclerosis in children came from the Bogalusa Heart Study in 1986, which...
9.
Stallings V, CORTNER J, Shannon B, Greene G, Collins S, Berman M, et al.
Am J Health Promot
. 1993 Oct;
8(2):106-8.
PMID: 10146825
No abstract available.
10.
Familial combined hyperlipidemia in children: clinical expression, metabolic defects, and management
CORTNER J, Coates P, Liacouras C, Jarvik G
J Pediatr
. 1993 Aug;
123(2):177-84.
PMID: 8345411
Familial combined hyperlipidemia (FCHL) is a dominantly inherited hyperlipidemia that occurs in at least 1% of the adult population and is responsible for 10% of premature coronary artery disease. In...