Iara De Schoenmacker
Overview
Explore the profile of Iara De Schoenmacker including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
11
Citations
35
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Sandy-Hindmarch O, Chang P, Scheuren P, De Schoenmacker I, Hubli M, Loizou C, et al.
Pain
. 2024 Nov;
PMID: 39588776
Focal nerve injuries are often associated with neuropathic pain. Preclinical research suggests altered neuroimmune signalling underlies such neuropathic pain; however, its cause remains poorly understood in humans. In this multicentre...
2.
De Schoenmacker I, Scheuren P, Sirucek L, Lutolf R, Gorrell L, Rosner J, et al.
Sci Rep
. 2024 Sep;
14(1):22070.
PMID: 39333624
Although reduced experimental pain habituation is proposed as a proxy of diminished endogenous pain modulatory capacity in chronic pain, prior studies show contradictory findings. Even across healthy participants, pain habituation...
3.
De Schoenmacker I, Costa Marques D, Scheuren P, Lutolf R, Gorrell L, Mehli S, et al.
Clin Neurophysiol
. 2024 Aug;
166:31-42.
PMID: 39094528
Objective: The present study aimed to investigate whether subjective and objective measures of pain habituation can be used as potential markers for central sensitization across various chronic pain patients. Methods:...
4.
Sirucek L, De Schoenmacker I, Scheuren P, Lutolf R, Gorrell L, Langenfeld A, et al.
Pain Rep
. 2024 Jun;
9(4):e1166.
PMID: 38910867
Introduction: In 85% of patients with chronic low back pain (CLBP), no specific pathoanatomical cause can be identified. Besides primary peripheral drivers within the lower back, spinal or supraspinal sensitization...
5.
Allmendinger F, Scheuren P, De Schoenmacker I, Brunner F, Rosner J, Curt A, et al.
J Pain Res
. 2024 Mar;
17:989-1003.
PMID: 38505501
Purpose: The pathophysiological mechanisms underlying the development of chronic pain in complex regional pain syndrome (CRPS) are diverse and involve both peripheral and central changes in pain processing, such as...
6.
De Schoenmacker I, Sirucek L, Scheuren P, Lutolf R, Gorrell L, Brunner F, et al.
Pain Rep
. 2023 Nov;
8(6):e1110.
PMID: 38027464
Introduction: First-line pain treatment is unsatisfactory in more than 50% of chronic pain patients, likely because of the heterogeneity of mechanisms underlying pain chronification. Objectives: This cross-sectional study aimed to...
7.
De Schoenmacker I, Mollo A, Scheuren P, Sirucek L, Brunner F, Schweinhardt P, et al.
Pain Med
. 2023 Mar;
24(8):974-984.
PMID: 36946277
Objective: Widespread pain hypersensitivity and enhanced temporal summation of pain (TSP) are commonly reported in patients with complex regional pain syndrome (CRPS) and discussed as proxies for central sensitization. This...
8.
Scheuren P, De Schoenmacker I, Rosner J, Brunner F, Curt A, Hubli M
Eur J Pain
. 2022 Sep;
27(1):72-85.
PMID: 36130736
Background: Allodynia and hyperalgesia are common signs in individuals with complex regional pain syndrome (CRPS), mainly attributed to sensitization of the nociceptive system. Appropriate diagnostic tools for the objective assessment...
9.
Lutolf R, De Schoenmacker I, Rosner J, Sirucek L, Schweinhardt P, Curt A, et al.
Eur J Pain
. 2022 Aug;
26(10):2176-2187.
PMID: 36000307
Background: Deficient endogenous pain modulation and increased nociceptive excitability are key features of central sensitization and can be assessed in humans by conditioned pain modulation (CPM, anti-nociceptive) and temporal summation...
10.
De Schoenmacker I, Leu C, Curt A, Hubli M
Eur J Pain
. 2022 Jun;
26(8):1679-1690.
PMID: 35671124
Background: Habituation is a response decrement resulting from repeated stimuli. Reduced habituation to noxious stimuli is considered to be a proxy for central sensitization in subjects with chronic pain. Despite...