Hugo Vanderstichele
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Explore the profile of Hugo Vanderstichele including associated specialties, affiliations and a list of published articles.
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89
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5012
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Recent Articles
11.
Konijnenberg E, Tomassen J, den Braber A, Ten Kate M, Yaqub M, Mulder S, et al.
Ann Neurol
. 2021 Feb;
89(5):987-1000.
PMID: 33583080
Objective: The present work was undertaken to study the genetic contribution to the start of Alzheimer's disease (AD) with amyloid and tau biomarkers in cognitively intact older identical twins. Methods:...
12.
Kumar S, Jagannath S, Francois C, Vanderstichele H, Stoops E, Lashuel H
Neurobiol Dis
. 2020 Sep;
146:105086.
PMID: 32971232
Increasing evidence suggests that alpha-synuclein (α-syn) oligomers are obligate intermediates in the pathway involved in α-syn fibrillization and Lewy body (LB) formation, and may also accumulate within LBs in Parkinson's...
13.
Boulo S, Kuhlmann J, Andreasson U, Brix B, Venkataraman I, Herbst V, et al.
Alzheimers Dement
. 2020 Aug;
16(11):1493-1503.
PMID: 32755010
Introduction: Reference materials based on human cerebrospinal fluid were certified for the mass concentration of amyloid beta (Aβ)1-42 (Aβ ). They are intended to be used to calibrate diagnostic assays...
14.
Mouton-Liger F, Dumurgier J, Cognat E, Hourregue C, Zetterberg H, Vanderstichele H, et al.
Alzheimers Res Ther
. 2020 Jul;
12(1):88.
PMID: 32690068
Background: The presynaptic protein neuregulin1 (NRG1) is cleaved by beta-site APP cleaving enzyme 1 (BACE1) in a similar way as amyloid precursor protein (APP) NRG1 can activate post-synaptic receptor tyrosine-protein...
15.
Candelise N, Schmitz M, Thune K, Cramm M, Rabano A, Zafar S, et al.
Transl Neurodegener
. 2020 Jan;
9:5.
PMID: 31988747
Background: α-Synuclein is a small soluble protein, whose physiological function in the healthy brain is poorly understood. Intracellular inclusions of α-synuclein, referred to as Lewy bodies (LBs), are pathological hallmarks...
16.
Galasko D, Xiao M, Xu D, Smirnov D, Salmon D, Dewit N, et al.
Alzheimers Dement (N Y)
. 2019 Dec;
5:871-882.
PMID: 31853477
Introduction: Amyloid, Tau, and neurodegeneration biomarkers can stage Alzheimer's Disease (AD). Synaptic biomarkers may help track cognition. Methods: In cognitively normal controls, Mild Cognitive Impairment (MCI) and AD, we investigated...
17.
Illan-Gala I, Pegueroles J, Montal V, Alcolea D, Vilaplana E, Bejanin A, et al.
Ann Clin Transl Neurol
. 2019 Dec;
6(12):2518-2530.
PMID: 31789459
Objective: We aimed to investigate the relationship between cerebrospinal fluid levels (CSF) of amyloid precursor protein (APP)-derived peptides related to the amyloidogenic pathway, cortical thickness, neuropsychological performance, and cortical gene...
18.
Chatterjee P, Elmi M, Goozee K, Shah T, Sohrabi H, Dias C, et al.
J Alzheimers Dis
. 2019 Aug;
71(3):775-783.
PMID: 31424403
Background: Aberrant amyloid-β (Aβ) deposition in the brain occurs two decades prior to the manifestation of Alzheimer's disease (AD) clinical symptoms and therefore brain Aβ load measured using PET serves...
19.
Schaeverbeke J, Gille B, Adamczuk K, Vanderstichele H, Chassaing E, Bruffaerts R, et al.
J Neurochem
. 2019 Feb;
149(1):139-157.
PMID: 30720873
The main pathophysiological alterations of Alzheimer's disease (AD) include loss of neuronal and synaptic integrity, amyloidogenic processing, and neuroinflammation. Similar alterations can, however, also be observed in cognitively intact older...
20.
Hilal S, Ikram M, Verbeek M, Franco O, Stoops E, Vanderstichele H, et al.
Stroke
. 2018 Oct;
49(11):2692-2698.
PMID: 30355213
Background and Purpose- Inflammation is involved in the pathogenesis of large artery atherosclerosis, ischemic stroke, and Alzheimer dementia. However, the role of inflammation in cerebral small vessel disease and neurodegeneration...