H Kenny Kuo
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Explore the profile of H Kenny Kuo including associated specialties, affiliations and a list of published articles.
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5
Citations
116
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Recent Articles
1.
Krasich R, Wu S, Kuo H, Kreuzer K
DNA Repair (Amst)
. 2015 Mar;
28:48-59.
PMID: 25731940
Pathways for tolerating and repairing DNA-protein crosslinks (DPCs) are poorly defined. We used transposon mutagenesis and candidate gene approaches to identify DPC-hypersensitive Escherichia coli mutants. DPCs were induced by azacytidine...
2.
Kuo H, McMahan S, Rota C, Kohl K, Sekelsky J
Genetics
. 2014 Sep;
198(3):935-45.
PMID: 25205745
Several helicases function during repair of double-strand breaks and handling of blocked or stalled replication forks to promote pathways that prevent formation of crossovers. Among these are the Bloom syndrome...
3.
Andersen S, Kuo H, Savukoski D, Brodsky M, Sekelsky J
PLoS Genet
. 2011 Oct;
7(10):e1002315.
PMID: 22022278
DNA repair mechanisms in mitotically proliferating cells avoid generating crossovers, which can contribute to genome instability. Most models for the production of crossovers involve an intermediate with one or more...
4.
Kuo H, Krasich R, Bhagwat A, Kreuzer K
Mol Microbiol
. 2010 Sep;
78(3):686-700.
PMID: 20807197
Anticancer drug 5-azacytidine (aza-C) induces DNA-protein cross-links (DPCs) between cytosine methyltransferase and DNA as the drug inhibits methylation. We found that mutants defective in the tmRNA translational quality control system...
5.
Kuo H, Griffith J, Kreuzer K
Cancer Res
. 2007 Sep;
67(17):8248-54.
PMID: 17804739
5-Azacytidine (aza-C) and its derivatives are cytidine analogues used for leukemia chemotherapy. The primary effect of aza-C is the prohibition of cytosine methylation, which results in covalent methyltransferase-DNA (MTase-DNA) adducts...