Graeme J Stewart
Overview
Explore the profile of Graeme J Stewart including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
47
Citations
3120
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Taylor M, Sidiqi H, Hare J, Kwok F, Choi B, Lee D, et al.
Intern Med J
. 2022 Dec;
52(12):2046-2067.
PMID: 36478370
Amyloidosis is a collection of diseases caused by the misfolding of proteins that aggregate into insoluble amyloid fibrils and deposit in tissues. While these fibrils may aggregate to form insignificant...
2.
Law S, Gatt P, Schibeci S, McKay F, Vucic S, Hart P, et al.
Genes Immun
. 2021 Jun;
22(4):227-233.
PMID: 34163021
Although genetic and epidemiological evidence indicates vitamin D insufficiency contributes to multiple sclerosis (MS), and serum levels of vitamin D increase on treatment with cholecalciferol, recent metanalyses indicate that this...
3.
Ong L, Parnell G, Veale K, Stewart G, Liddle C, Booth D
Genes Immun
. 2020 Oct;
21(5):335-347.
PMID: 33037402
Multiple lines of evidence indicate Multiple Sclerosis (MS) is affected by vitamin D. This effect may be mediated by methylation in immune cell progenitors. We aimed to determine (1) if...
4.
Bart N, Thomas L, Korczyk D, Atherton J, Stewart G, Fatkin D
Heart Lung Circ
. 2020 Feb;
29(4):575-583.
PMID: 32001152
Amyloid cardiomyopathy is emerging as an important and under-recognised cause of heart failure and cardiac arrhythmias, especially in older adults. This disorder is characterised by extracellular deposition of amyloid fibrils...
5.
Afrasiabi A, Parnell G, Swaminathan S, Stewart G, Booth D
Sci Rep
. 2020 Jan;
10(1):193.
PMID: 31932685
Translating the findings of genome wide association studies (GWAS) to new therapies requires identification of the relevant immunological contexts to interrogate for genetic effects. In one of the largest GWAS,...
6.
Ong L, Parnell G, Afrasiabi A, Stewart G, Swaminathan S, Booth D
Genes Immun
. 2019 Oct;
21(2):91-99.
PMID: 31619767
Epstein-Barr Virus (EBV) infection appears to be necessary for the development of Multiple Sclerosis (MS), although the specific mechanisms are unknown. More than 200 single-nucleotide polymorphisms (SNPs) are known to...
7.
Afrasiabi A, Parnell G, Fewings N, Schibeci S, Basuki M, Chandramohan R, et al.
Genome Med
. 2019 May;
11(1):26.
PMID: 31039804
Background: Genome wide association studies have identified > 200 susceptibility loci accounting for much of the heritability of multiple sclerosis (MS). Epstein-Barr virus (EBV), a memory B cell tropic virus,...
8.
Parnell G, Schibeci S, Fewings N, Afrasiabi A, Law S, Samaranayake S, et al.
Hum Mol Genet
. 2018 Oct;
28(2):269-278.
PMID: 30285234
Epidemiological, molecular and genetic studies have indicated that high serum vitamin D levels are associated with lower risk of several autoimmune diseases. The vitamin D receptor (VDR) binding sites in...
9.
Sheean R, McKay F, Cretney E, Bye C, Perera N, Tomas D, et al.
JAMA Neurol
. 2018 Mar;
75(6):681-689.
PMID: 29507931
Importance: Neuroinflammation appears to be a key modulator of disease progression in amyotrophic lateral sclerosis (ALS) and thereby a promising therapeutic target. The CD4+Foxp3+ regulatory T-cells (Tregs) infiltrating into the...
10.
Booth D, Fewings N, Parnell G, McKay F, Stewart G
Mult Scler J Exp Transl Clin
. 2017 Jun;
2:2055217316637087.
PMID: 28607721
A promising new avenue of MS research that may lead to a better understanding of pathogenesis, progression and therapeutic response, and to development of new therapies, comes from the recent...