G T van der Horst
Overview
Explore the profile of G T van der Horst including associated specialties, affiliations and a list of published articles.
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Articles
38
Citations
2107
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Recent Articles
1.
Brash D, Wikonkal N, Remenyik E, van der Horst G, Friedberg E, Cheo D, et al.
J Invest Dermatol
. 2001 Nov;
117(5):1234-40.
PMID: 11710938
The stratum corneum and DNA repair do not completely protect keratinocytes from ultraviolet B. A third defense prevents cells with DNA photoproducts from becoming precancerous mutant cells: apoptosis of ultraviolet-damaged...
2.
Murai M, Enokido Y, Inamura N, Yoshino M, Nakatsu Y, van der Horst G, et al.
Proc Natl Acad Sci U S A
. 2001 Nov;
98(23):13379-84.
PMID: 11687625
Xeroderma pigmentosum (XP) and Cockayne syndrome (CS) are rare autosomal recessive disorders associated with a defect in the nucleotide excision repair (NER) pathway required for the removal of DNA damage...
3.
Boonstra A, van Oudenaren A, Baert M, van Steeg H, Leenen P, van der Horst G, et al.
J Invest Dermatol
. 2001 Jul;
117(1):141-6.
PMID: 11442761
Ultraviolet B irradiation has serious consequences for cellular immunity and can suppress the rejection of skin tumors and the resistance to infectious diseases. DNA damage plays a crucial role in...
4.
Yagita K, Tamanini F, van der Horst G, Okamura H
Science
. 2001 Apr;
292(5515):278-81.
PMID: 11303101
In mammals, the central circadian pacemaker resides in the hypothalamic suprachiasmatic nucleus (SCN), but circadian oscillators also exist in peripheral tissues. Here, using wild-type and cryptochrome (mCry)-deficient cell lines derived...
5.
Daan S, Albrecht U, van der Horst G, Illnerova H, Roenneberg T, Wehr T, et al.
J Biol Rhythms
. 2001 Apr;
16(2):105-16.
PMID: 11302553
The hypothesis is advanced that the circadian pacemaker in the mammalian suprachiasmatic nucleus (SCN) is composed at the molecular level of a nonredundant double complex of circadian genes (per1, cry1,...
6.
Lu Y, Lian H, Sharma P, Schreiber-Agus N, Russell R, Chin L, et al.
Mol Cell Biol
. 2001 Mar;
21(5):1810-8.
PMID: 11238917
Cells isolated from individuals with Cockayne syndrome (CS) have a defect in transcription-coupled DNA repair, which rapidly corrects certain DNA lesions located on the transcribed strand of active genes. Despite...
7.
Yamaguchi S, Kobayashi M, Mitsui S, Ishida Y, van der Horst G, Suzuki M, et al.
Nature
. 2001 Feb;
409(6821):684.
PMID: 11217850
No abstract available.
8.
Wijnhoven S, Kool H, van Oostrom C, Beems R, Mullenders L, van Zeeland A, et al.
Cancer Res
. 2000 Nov;
60(20):5681-7.
PMID: 11059760
Cockayne syndrome (CS) patients are deficient in the transcription coupled repair (TCR) subpathway of nucleotide excision repair (NER) but in contrast to xeroderma pigmentosum patients, who have a defect in...
9.
Wijnhoven S, Kool H, Mullenders L, van Zeeland A, Friedberg E, van der Horst G, et al.
Oncogene
. 2000 Oct;
19(43):5034-7.
PMID: 11042691
DNA damages caused by cellular metabolites and environmental agents induce mutations, that may predispose to cancer. Nucleotide excision repair (NER) is a major cellular defence mechanism acting on a variety...
10.
van Oosten M, Rebel H, Friedberg E, van Steeg H, van der Horst G, van Kranen H, et al.
Proc Natl Acad Sci U S A
. 2000 Sep;
97(21):11268-73.
PMID: 11005836
Nucleotide excision repair (NER), apoptosis, and cell-cycle regulation are major defense mechanisms against the carcinogenic effects of UVB light. NER eliminates UVB-induced DNA photolesions via two subpathways: global genome repair...