Folkert J Ten Cate
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Explore the profile of Folkert J Ten Cate including associated specialties, affiliations and a list of published articles.
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3591
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Recent Articles
11.
van Dijk S, Boontje N, Heymans M, Ten Cate F, Michels M, Dos Remedios C, et al.
Pflugers Arch
. 2013 Nov;
466(8):1619-33.
PMID: 24186209
Mutations in the MYBPC3 gene, encoding cardiac myosin binding protein C (cMyBP-C) are frequent causes of hypertrophic cardiomyopathy (HCM). Previously, we have presented evidence for reduced cMyBP-C expression (haploinsufficiency), in...
12.
Kuster D, Mulders J, Ten Cate F, Michels M, Dos Remedios C, da Costa Martins P, et al.
J Mol Cell Cardiol
. 2013 Oct;
65:59-66.
PMID: 24083979
Hypertrophic cardiomyopathy (HCM) is predominantly caused by mutations in genes encoding sarcomeric proteins. One of the most frequent affected genes is MYBPC3, which encodes the thick filament protein cardiac myosin...
13.
Vriesendorp P, Schinkel A, Van Cleemput J, Willems R, Jordaens L, Theuns D, et al.
Am Heart J
. 2013 Sep;
166(3):496-502.
PMID: 24016499
Background: Sudden cardiac death (SCD) is the most devastating complication of hypertrophic cardiomyopathy (HCM), but this can be prevented by an implantable cardioverter-defibrillator (ICD). The aim of this study is...
14.
Witjas-Paalberends E, Piroddi N, Stam K, van Dijk S, Oliviera V, Ferrara C, et al.
Cardiovasc Res
. 2013 May;
99(3):432-41.
PMID: 23674513
Aims: Familial hypertrophic cardiomyopathy (HCM), frequently caused by sarcomeric gene mutations, is characterized by cellular dysfunction and asymmetric left-ventricular (LV) hypertrophy. We studied whether cellular dysfunction is due to an...
15.
Sequeira V, Wijnker P, Nijenkamp L, Kuster D, Najafi A, Witjas-Paalberends E, et al.
Circ Res
. 2013 Mar;
112(11):1491-505.
PMID: 23508784
Rationale: High-myofilament Ca(2+) sensitivity has been proposed as a trigger of disease pathogenesis in familial hypertrophic cardiomyopathy (HCM) on the basis of in vitro and transgenic mice studies. However, myofilament...
16.
Soliman O, Geleijnse M, Caliskan K, Vletter W, Ten Cate F
Expert Opin Med Diagn
. 2013 Mar;
6(3):221-33.
PMID: 23480687
Background: Noncompaction cardiomyopathy (NCCM) is a new pathoanatomic entity, disputably believed to result from abnormal arrest in embryonic endomyocardial morphogenesis. During almost three decades of research of NCCM, more knowledge...
17.
van Dalen B, Tzikas A, Soliman O, Heuvelman H, Vletter W, Ten Cate F, et al.
Echocardiography
. 2013 Jan;
30(3):293-300.
PMID: 23347129
Background: Angina and an electrocardiographic strain pattern are potential manifestations of subendocardial ischemia in aortic stenosis (AS). Left ventricular (LV) twist is known to increase proportionally to the severity of...
18.
Ren B, de Groot-de Laat L, McGhie J, Vletter W, Ten Cate F, Geleijnse M
J Am Soc Echocardiogr
. 2012 Dec;
26(3):261-9.
PMID: 23228655
Background: The aim of this study was to estimate geometric errors made by the two-dimensional (2D) transthoracic echocardiographic (TTE) pulsed-wave Doppler flow (PWDF) method in calculating regurgitant volume (RVol) and...
19.
Kauer F, van Dalen B, Michels M, Soliman O, Vletter W, van Slegtenhorst M, et al.
Echocardiography
. 2012 Dec;
30(5):558-63.
PMID: 23228071
Background: Tissue Doppler imaging (TDI) of the mitral annulus has been proposed as an alternative for the identification of hypertrophic cardiomyopathy (HCM) genetically affected subjects without left ventricular hypertrophy (G+/LVH-)....
20.
Kauer F, van Dalen B, Soliman O, der Zwaan H, Vletter W, Schinkel A, et al.
Eur Heart J Cardiovasc Imaging
. 2012 Aug;
14(5):435-42.
PMID: 22898715
Aims: This study sought to investigate regional left ventricular (LV) rotation in patients with hypertrophic cardiomyopathy (HCM). Methods And Results: The study comprised 44 patients with HCM with a typical...