Elza Muscelli
Overview
Explore the profile of Elza Muscelli including associated specialties, affiliations and a list of published articles.
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Articles
39
Citations
1629
Followers
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Recent Articles
1.
Trico D, Rebelos E, Astiarraga B, Baldi S, Scozzaro T, Sacchetta L, et al.
J Clin Endocrinol Metab
. 2024 Apr;
110(3):e667-e674.
PMID: 38635405
Context: Hypertriglyceridemia is a risk factor for developing type 2 diabetes (T2D) and might contribute to its pathogenesis either directly or through elevation of nonesterified fatty acids (NEFAs). Objective: This...
2.
Grespan E, Guolo A, Muscelli E, Ferrannini E, Mari A
J Clin Endocrinol Metab
. 2022 Apr;
107(7):2092-2100.
PMID: 35397169
Context: Loss of the incretin effect (IE) in type 2 diabetes (T2D) contributes to hyperglycemia and the mechanisms underlying this impairment are unclear. Objective: To quantify the IE impairment in...
3.
Breder I, Wolf V, Soares A, de Carvalho L, Kimura-Medorima S, Cintra R, et al.
Diabetes Metab
. 2021 Nov;
48(2):101304.
PMID: 34808345
No abstract available.
4.
Bizzotto R, Trico D, Natali A, Gastaldelli A, Muscelli E, De Fronzo R, et al.
Diabetes Care
. 2021 Aug;
44(9):2115-2123.
PMID: 34362813
Objective: Endogenous insulin clearance (EIC) is physiologically reduced at increasing insulin secretion rate (ISR). Computing EIC at the prevailing ISR does not distinguish the effects of hypersecretion from those of...
5.
Wolf V, Breder I, de Carvalho L, Soares A, Cintra R, Barreto J, et al.
Nutr Diabetes
. 2021 Jun;
11(1):17.
PMID: 34120150
We compared the effect of dapagliflozin versus glibenclamide on the ratio of lean-to total mass in patients with type 2 diabetes mellitus, carotid subclinical atherosclerosis, HbA1c 7.0-9.0% and 40-70 years-old....
6.
Chueire V, Muscelli E
Arch Endocrinol Metab
. 2020 Dec;
65(1):24-31.
PMID: 33320449
Deleterious effects of free fatty acids, FFAs, on insulin sensitivity are observed in vivo studies in humans. Mechanisms include impaired insulin signaling, oxidative stress, inflammation, and mitochondrial dysfunction, but the...
7.
Breder I, Breder J, Bonilha I, Munhoz D, Medorima S, Oliveira D, et al.
Ther Adv Chronic Dis
. 2020 Oct;
11:2040622320959248.
PMID: 33062236
Background: Patients with type 2 diabetes mellitus (T2DM) remain at increased cardiovascular residual risk and endothelial dysfunction, even after optimizing metabolic control and treatment by sodium-glucose-2 transporter inhibitors (SGLT2-is). The...
8.
Ferrannini E, Murthy A, Lee Y, Muscelli E, Weiss S, Ostroff R, et al.
Diabetes Care
. 2020 Jun;
43(9):2183-2189.
PMID: 32527800
Objective: To assess the effects of empagliflozin, a selective sodium-glucose cotransporter 2 (SGLT2) inhibitor, on broad biological systems through proteomics. Research Design And Methods: Aptamer-based proteomics was used to quantify...
9.
Ferrannini E, Pereira-Moreira R, Seghieri M, Rebelos E, Souza A, Chueire V, et al.
BMJ Open Diabetes Res Care
. 2020 May;
8(1).
PMID: 32423964
Introduction: Insulin regulates renal glucose production and utilization; both these fluxes are increased in type 2 diabetes (T2D). Whether insulin also controls urinary glucose excretion is not known. Methods: We...
10.
Pereira-Moreira R, Muscelli E
J Diabetes Res
. 2020 May;
2020:8492467.
PMID: 32377524
Renal proximal tubules reabsorb glucose from the glomerular filtrate and release it back into the circulation. Modulation of glomerular filtration and renal glucose disposal are some of the insulin actions,...