Dmitry V Bulavin
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Explore the profile of Dmitry V Bulavin including associated specialties, affiliations and a list of published articles.
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5501
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Recent Articles
1.
Triana-Martinez F, Pierantoni A, Graca D, Bergo V, Emelyanov A, Grigorash B, et al.
bioRxiv
. 2024 Jul;
PMID: 39026790
The ability of an organism to overcome infectious diseases has traditionally been linked to killing invading pathogens. Accumulating evidence, however, indicates that, apart from restricting pathogen loads, organismal survival is...
2.
Grigorash B, van Essen D, Liang G, Grosse L, Emelyanov A, Kang Z, et al.
Nat Cell Biol
. 2023 Aug;
25(9):1265-1278.
PMID: 37652981
Despite advances in four-factor (4F)-induced reprogramming (4FR) in vitro and in vivo, how 4FR interconnects with senescence remains largely under investigated. Here, using genetic and chemical approaches to manipulate senescent...
3.
Born E, Lipskaia L, Breau M, Houssaini A, Beaulieu D, Marcos E, et al.
Circulation
. 2022 Dec;
147(8):650-666.
PMID: 36515093
Background: Senescent cells (SCs) are involved in proliferative disorders, but their role in pulmonary hypertension remains undefined. We investigated SCs in patients with pulmonary arterial hypertension and the role of...
4.
Grosse L, Bulavin D
Aging (Albany NY)
. 2020 Jun;
12(11):11152-11160.
PMID: 32535553
Data obtained from genetically modified mouse models suggest a detrimental role for p16 senescent cells in physiological aging and age-related pathologies. Our recent analysis of aging mice revealed a continuous...
5.
Grosse L, Wagner N, Emelyanov A, Molina C, Lacas-Gervais S, Wagner K, et al.
Cell Metab
. 2020 Jun;
32(1):87-99.e6.
PMID: 32485135
The accumulation of senescent cells can drive many age-associated phenotypes and pathologies. Consequently, it has been proposed that removing senescent cells might extend lifespan. Here, we generated two knockin mouse...
6.
Filipponi D, Emelyanov A, Muller J, Molina C, Nichols J, Bulavin D
Mol Cell
. 2019 Apr;
74(4):651-663.e8.
PMID: 30954402
Accumulating evidence supports the role of the DNA damage response (DDR) in the negative regulation of tumorigenesis. Here, we found that DDR signaling poises a series of epigenetic events, resulting...
7.
Bertero T, Oldham W, Grasset E, Bourget I, Boulter E, Pisano S, et al.
Cell Metab
. 2018 Oct;
29(1):124-140.e10.
PMID: 30293773
Dysregulation of extracellular matrix (ECM) deposition and cellular metabolism promotes tumor aggressiveness by sustaining the activity of key growth, invasion, and survival pathways. Yet mechanisms by which biophysical properties of...
8.
Grasset E, Bertero T, Bozec A, Friard J, Bourget I, Pisano S, et al.
Cancer Res
. 2018 Jul;
78(18):5229-5242.
PMID: 30026329
In squamous cell carcinoma (SCC), tissue invasion by collectively invading cells requires physical forces applied by tumor cells on their surrounding extracellular matrix (ECM). Cancer-related ECM is composed of thick...
9.
Brichkina A, Bulavin D
Oncotarget
. 2017 Feb;
8(9):14275-14276.
PMID: 28199960
No abstract available.
10.
Brichkina A, Bertero T, Loh H, Nguyen N, Emelyanov A, Rigade S, et al.
Genes Dev
. 2016 Dec;
30(23):2623-2636.
PMID: 28007785
Expansion of neoplastic lesions generates the initial signal that instigates the creation of a tumor niche. Nontransformed cell types within the microenvironment continuously coevolve with tumor cells to promote tumorigenesis....