David M Brass
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Explore the profile of David M Brass including associated specialties, affiliations and a list of published articles.
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36
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1248
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Recent Articles
21.
Brass D, Yang I, Kennedy M, Whitehead G, Rutledge H, Burch L, et al.
Immunogenetics
. 2008 Jun;
60(7):353-69.
PMID: 18553078
Chronic LPS inhalation causes submucosal thickening and airway narrowing. To address the hypothesis that environmental airway disease is, in part, a fibroproliferative lung disease, we exposed C57BL/6 mice daily to...
22.
Brass D, Hollingsworth J, Cinque M, Li Z, Potts E, Toloza E, et al.
Am J Respir Cell Mol Biol
. 2008 Jun;
39(5):584-90.
PMID: 18539952
Lipopolysaccharide (LPS) is ubiquitous in the environment. Recent epidemiologic data suggest that occupational exposure to inhaled LPS can contribute to the progression of chronic obstructive pulmonary disease. To address the...
23.
Hollingsworth J, Maruoka S, Li Z, Potts E, Brass D, Garantziotis S, et al.
J Immunol
. 2007 Sep;
179(7):4367-75.
PMID: 17878331
Exposure to ozone in air pollution in urban environments is associated with increases in pulmonary-related hospitalizations and mortality. Because ozone also alters clearance of pulmonary bacterial pathogens, we hypothesized that...
24.
Brass D, Hollingsworth J, Fessler M, Savov J, Maxwell A, Whitehead G, et al.
J Allergy Clin Immunol
. 2007 May;
120(1):121-7.
PMID: 17512577
Background: The contribution of IL-1beta signaling through the IL-1 type 1 receptor (IL-1R1) to the development of persistent LPS-induced airway disease has not been investigated. Objective: To determine the importance...
25.
Hollingsworth J, Li Z, Brass D, Garantziotis S, Timberlake S, Kim A, et al.
Am J Respir Cell Mol Biol
. 2007 Apr;
37(2):248-53.
PMID: 17446529
LPS from bacteria is ubiquitous in the environment and can cause airway disease and modify allergic asthma. Identification of gene products that modulate the biologic response to inhaled LPS will...
26.
Brass D, Hollingsworth J, McElvania-TeKippe E, Garantziotis S, Hossain I, Schwartz D
Am J Physiol Lung Cell Mol Physiol
. 2007 Mar;
293(1):L77-83.
PMID: 17384086
Chronic lipopolysaccharide (LPS) inhalation in rodents recapitulates many classic features of chronic obstructive pulmonary disease seen in humans, including airways hyperresponsiveness, neutrophilic inflammation, cytokine production in the lung, and small...
27.
Brass D, Tomfohr J, Yang I, Schwartz D
Proc Am Thorac Soc
. 2007 Jan;
4(1):92-100.
PMID: 17202297
Idiopathic interstitial pneumonia represents a broad category of lung disorders characterized by scarring or fibrosis of the lung accompanied by varying degrees of inflammation. A number of important hypotheses based...
28.
Leukocyte-derived IL-10 reduces subepithelial fibrosis associated with chronically inhaled endotoxin
Garantziotis S, Brass D, Savov J, Hollingsworth J, McElvania-TeKippe E, Berman K, et al.
Am J Respir Cell Mol Biol
. 2006 Jul;
35(6):662-7.
PMID: 16809636
Endotoxin (LPS), a Gram-negative cell wall component, has potent proinflammatory properties. Acute LPS exposure causes airway inflammation; chronic exposure causes airway hyperreactivity and remodeling. IL-10 is an important antiinflammatory cytokine,...
29.
Savov J, Brass D, Lawson B, McElvania-TeKippe E, Walker J, Schwartz D
Am J Physiol Lung Cell Mol Physiol
. 2005 Apr;
289(2):L329-37.
PMID: 15833764
Although chronic inhalation of endotoxin or lipopolysaccharide (LPS) causes all of the classic features of asthma, including airway hyperreactivity, airway inflammation, and airway remodeling, the mechanisms involved in this process...
30.
Hollingsworth J, Chen B, Brass D, Berman K, Gunn M, Cook D, et al.
Am J Respir Crit Care Med
. 2004 Dec;
171(8):806-13.
PMID: 15618460
Rapid and selective recruitment of neutrophils into the airspace in response to LPS facilitates the clearance of bacterial pathogens. However, neutrophil infiltration can also participate in the development and progression...