Daniel B Metzger
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Explore the profile of Daniel B Metzger including associated specialties, affiliations and a list of published articles.
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14
Citations
109
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Recent Articles
1.
Davis D, Metzger D, Vann P, Wong J, Shetty R, Forster M, et al.
Psychopharmacology (Berl)
. 2023 May;
240(6):1343-1358.
PMID: 37127834
Recreational and medical use of stimulants among young adults have gained popularity in the United States over the last decade and their use may increase vulnerability to brain biochemical changes...
2.
Davis D, Metzger D, Vann P, Wong J, Subasinghe K, Garlotte I, et al.
Psychopharmacology (Berl)
. 2022 Mar;
239(7):2331-2349.
PMID: 35347365
Rationale: Recreational and medical use of stimulants is increasing, and their use may increase susceptibility to aging and promote neurobehavioral impairments. The long-term consequences of these psychostimulants and how they...
3.
Ryou M, Chen X, Cai M, Wang H, Jung M, Metzger D, et al.
Front Aging Neurosci
. 2021 Jul;
13:674688.
PMID: 34276338
In mouse models of Alzheimer's disease (AD), normobaric intermittent hypoxia training (IHT) can preserve neurobehavioral function when applied before deficits develop, but IHT's effectiveness after onset of amyloid-β (Aβ) accumulation...
4.
Jung M, Metzger D, Hall J
Pharmacol Biochem Behav
. 2020 Feb;
191:172873.
PMID: 32105662
Many elderly American women use CNS depressant benzodiazepine (BZD) to ameliorate anxiety or insomnia. However, the chronic use of BZD (cBZD) is prevalent, causing adverse effects of BZD that include...
5.
Tan S, Metzger D, Jung M
Pharmacol Biochem Behav
. 2018 Jul;
172:59-67.
PMID: 30030126
Benzodiazepine (BZD) is a commonly prescribed anxiolytic and sedation aid medication, especially in elderly women. However, long-term use of BZD provokes adverse nontherapeutic effects that include movement deficit. Here, we...
6.
Ryou M, Mallet R, Metzger D, Jung M
Am J Physiol Regul Integr Comp Physiol
. 2017 May;
313(1):R10-R18.
PMID: 28490448
Abrupt cessation of chronic alcohol consumption triggers signaling cascades that harm vulnerable brain regions and produce neurobehavioral deficits. We have demonstrated that a program of intermittent, normobaric hypoxia training (IHT)...
7.
Jung M, Metzger D
Behav Brain Res
. 2016 Aug;
314:199-214.
PMID: 27503149
Ethanol withdrawal (EW) is referred to the abrupt termination of long-term heavy drinking, and provokes oxidative brain damage. Here, we investigated whether the cerebellum and hippocampus of female rats are...
8.
Jung M, Metzger D, Das H
J Pharmacol Exp Ther
. 2016 Jun;
358(3):516-26.
PMID: 27278235
Presenilin-1 (PS1) is a core component of γ-secretase that is involved in neurodegeneration. We have previously shown that PS1 interacts with a mitogen-activated protein kinase [(MAPK) jun-NH2-terminal-kinase], and another MAPK...
9.
Jung M, Metzger D
J Pharmacol Exp Ther
. 2014 Nov;
352(2):258-66.
PMID: 25406171
The acetylation of histone proteins in the core of DNA regulates gene expression, including those affecting mitochondria. Both histone acetylation and mitochondrial deficit have been implicated in neuronal damage associated...
10.
Ju X, Mallet R, Downey H, Metzger D, Jung M
J Appl Physiol (1985)
. 2012 Mar;
112(10):1706-14.
PMID: 22403345
Intermittent hypoxia (IH) conditioning minimizes neurocognitive impairment and stabilizes brain mitochondrial integrity during ethanol withdrawal (EW) in rats, but the mitoprotective mechanism is unclear. We investigated whether IH conditioning protects...