Daniel A Goodenough
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Explore the profile of Daniel A Goodenough including associated specialties, affiliations and a list of published articles.
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Recent Articles
1.
Chai Z, Goodenough D, Paul D
Mol Biol Cell
. 2011 Oct;
22(23):4503-12.
PMID: 21965293
The three connexins expressed in the ocular lens each contain PDZ domain-binding motifs directing a physical association with the scaffolding protein ZO-1, but the significance of the interaction is unknown....
2.
Magnotti L, Goodenough D, Paul D
Glia
. 2011 May;
59(7):1064-74.
PMID: 21538560
CNS glia exhibit a variety of gap junctional interactions: between neighboring astrocytes, between neighboring oligodendrocytes, between astrocytes and oligodendrocytes, and as 'reflexive' structures between layers of myelin in oligodendrocytes. Together,...
3.
Magnotti L, Goodenough D, Paul D
Glia
. 2010 Nov;
59(1):26-34.
PMID: 21046554
Human genetic diseases and mouse knockouts illustrate that the maintenance of central nervous system myelin requires connexin expression by both astrocytes and oligodendrocytes. Because these cell types express nonoverlapping sets...
4.
Hou J, Goodenough D
Curr Opin Nephrol Hypertens
. 2010 Jul;
19(5):483-8.
PMID: 20616717
Purpose Of Review: Claudin-16 and claudin-19 play a major role in the regulation of magnesium reabsorption in the thick ascending limb (TAL). This review describes recent findings of the physiological...
5.
Goodenough D, Paul D
Cold Spring Harb Perspect Biol
. 2010 Jan;
1(1):a002576.
PMID: 20066080
Gap junctions are aggregates of intercellular channels that permit direct cell-cell transfer of ions and small molecules. Initially described as low-resistance ion pathways joining excitable cells (nerve and muscle), gap...
6.
Hou J, Renigunta A, Gomes A, Hou M, Paul D, Waldegger S, et al.
Proc Natl Acad Sci U S A
. 2009 Aug;
106(36):15350-5.
PMID: 19706394
Claudins are tight junction integral membrane proteins that are key regulators of the paracellular pathway. Defects in claudin-16 (CLDN16) and CLDN19 function result in the inherited human renal disorder familial...
7.
Shan Q, Himmerkus N, Hou J, Goodenough D, Bleich M
Ann N Y Acad Sci
. 2009 Jun;
1165:148-51.
PMID: 19538300
Tight junction (TJ) properties are determined by membrane protein complexes of neighboring cells that form both a barrier and a selective pathway for paracellular substrate transport. Our previous work supports...
8.
Calera M, Wang Z, Sanchez-Olea R, Paul D, Civan M, Goodenough D
Invest Ophthalmol Vis Sci
. 2009 Jan;
50(5):2185-93.
PMID: 19168903
Purpose: Conditional inactivation of connexin43 (Cx43) in the pigmented epithelium of the mouse eye results in a reduction in aqueous humor production and complete loss of the vitreous chamber. It...
9.
Himmerkus N, Shan Q, Goerke B, Hou J, Goodenough D, Bleich M
Am J Physiol Renal Physiol
. 2008 Sep;
295(6):F1641-7.
PMID: 18784260
Claudin-16 is defective in familial hypomagnesemia with hypercalciuria and nephrocalcinosis (FHHNC). Claudin-16 knockdown (CLDN16 KD) mice show reduced cation selectivity in the thick ascending limb. The defect leads to a...
10.
Hou J, Renigunta A, Konrad M, Gomes A, Schneeberger E, Paul D, et al.
J Clin Invest
. 2008 Jan;
118(2):619-28.
PMID: 18188451
Tight junctions (TJs) play a key role in mediating paracellular ion reabsorption in the kidney. Familial hypomagnesemia with hypercalciuria and nephrocalcinosis (FHHNC) is an inherited disorder caused by mutations in...