D J Stearns-Kurosawa
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Explore the profile of D J Stearns-Kurosawa including associated specialties, affiliations and a list of published articles.
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Articles
23
Citations
709
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Recent Articles
1.
Hall G, Kurosawa S, Stearns-Kurosawa D
Methods Mol Biol
. 2021 Mar;
2291:353-364.
PMID: 33704763
Previous methods of infecting mice with Shiga toxin-producing E. coli (STEC) required suppression of host immune function or ablation of the gut microbiota to induce susceptibility to gastrointestinal colonization. Consequently,...
2.
Feng F, Thompson M, Thomas B, Duffy E, Kim J, Kurosawa S, et al.
PLoS One
. 2019 Apr;
14(4):e0209060.
PMID: 30995241
Biomarkers are fundamental to basic and clinical research outcomes by reporting host responses and providing insight into disease pathophysiology. Measuring biomarkers with research-use ELISA kits is universal, yet lack of...
3.
Hall G, Kurosawa S, Stearns-Kurosawa D
Infect Immun
. 2018 Aug;
86(11).
PMID: 30150257
Shiga toxin-producing (STEC) bacteria are globally important gastrointestinal pathogens causing hemorrhagic gastroenteritis with variable progression to potentially fatal Shiga toxicosis. Little is known about the potential effects of derived Shiga-like...
4.
Lee B, Mayer C, Leibowitz C, Stearns-Kurosawa D, Kurosawa S
Blood
. 2013 Jun;
122(5):803-6.
PMID: 23733336
Enterohemorrhagic Escherichia coli (EHEC) produce ribosome-inactivating Shiga toxins (Stx1, Stx2) responsible for development of hemolytic uremic syndrome (HUS) and acute kidney injury (AKI). Some patients show complement activation during EHEC...
5.
Stearns-Kurosawa D, Collins V, Freeman S, Tesh V, Kurosawa S
Infect Immun
. 2010 Mar;
78(6):2497-504.
PMID: 20308301
Shiga toxin-producing Escherichia coli is a principal source of regional outbreaks of bloody diarrhea and hemolytic-uremic syndrome in the United States and worldwide. Primary bacterial virulence factors are Shiga toxin...
6.
Reiner A, Carty C, Jenny N, Nievergelt C, Cushman M, Stearns-Kurosawa D, et al.
J Thromb Haemost
. 2008 Aug;
6(10):1625-32.
PMID: 18680534
Background And Objectives: Genes encoding protein C anticoagulant pathways are candidates for atherothrombotic and other aging-related disorders. Methods: Using a tagSNP approach, and data from the Cardiovascular Health Study (CHS),...
7.
Ireland H, Konstantoulas C, Cooper J, Hawe E, Humphries S, Mather H, et al.
Atherosclerosis
. 2005 Jun;
183(2):283-92.
PMID: 15921688
We have progressively analysed three studies of coronary heart disease (CHD) for a variant in EPCR (Ser219Gly). Initially, in a prospective study, NPHSII, while no overall CHD-risk was identified in...
8.
Stearns-Kurosawa D, Burgin C, Parker D, Comp P, Kurosawa S
J Thromb Haemost
. 2003 Jul;
1(4):855-6.
PMID: 12871429
No abstract available.
9.
Kaneko T, Stearns-Kurosawa D, Taylor Jr F, Twigg M, Osaki K, Kinasewitz G, et al.
Shock
. 2003 Jul;
20(2):130-7.
PMID: 12865656
CD10, also known as neutral endopeptidase or CALLA, is a major metalloproteinase that regulates levels of biologically active peptides that initiate inflammatory, cardiovascular, and neurogenic responses. Relative tissue expression levels...
10.
Boomsma M, Stearns-Kurosawa D, Stegeman C, Raschi E, Meroni P, Kurosawa S, et al.
Clin Exp Immunol
. 2002 May;
128(1):187-94.
PMID: 12033194
Elevated soluble thrombomodulin (sTM) levels are an accepted marker of endothelial damage. The physiological significance of plasma endothelial protein C receptor (sEPCR) levels is not known. To assess the relevance...