Craig Zaidman
Overview
Explore the profile of Craig Zaidman including associated specialties, affiliations and a list of published articles.
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11
Citations
327
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Recent Articles
1.
Zhu C, Zaidman C, Youn B, Paradis A, Raynaud S, Neville B, et al.
J Comp Eff Res
. 2024 Jul;
:e230187.
PMID: 38963060
Nusinersen, administered by intrathecal injection at a dose of 12 mg, is indicated across all ages for the treatment of spinal muscular atrophy (SMA). Evidence on real-world healthcare resource use...
2.
Evesson F, Dziaduch G, Bryen S, Moore F, Pittman S, Devanapalli B, et al.
Hum Mol Genet
. 2023 Mar;
32(12):2084-2092.
PMID: 36920481
Recessive variants in the oxidoreductase PYROXD1 are reported to cause a myopathy in 22 affected individuals from 15 families. Here, we describe two female probands from unrelated families presenting with...
3.
Chand D, Zaidman C, Arya K, Millner R, Farrar M, Mackie F, et al.
J Pediatr
. 2020 Dec;
231:265-268.
PMID: 33259859
Spinal muscular atrophy is treated with onasemnogene abeparvovec, which replaces the missing survival motor neuron 1 gene via an adeno-associated virus vector. As of July 1, 2020, we had identified...
4.
Leitner M, Kapur K, Darras B, Yang M, Wong B, Dalle Pazze L, et al.
Ann Clin Transl Neurol
. 2019 Dec;
7(1):4-14.
PMID: 31876124
Objective: To evaluate the sensitivity of electrical impedance myography (EIM) to disease progression in both ambulatory and non-ambulatory boys with DMD. Methods And Participants: A non-blinded, longitudinal cohort study of...
5.
Wang D, Wu X, Bai Y, Zaidman C, Grider T, Kamholz J, et al.
Ann Clin Transl Neurol
. 2017 Apr;
4(4):236-245.
PMID: 28382305
Objective: To determine whether predicted fork stalling and template switching (FoSTeS) during mitosis deletes exon 4 in peripheral myelin protein 22 KD (PMP22) and causes gain-of-function mutation associated with peripheral...
6.
Rutkove S, Wu J, Zaidman C, Kapur K, Yim S, Pasternak A, et al.
Clin Neurophysiol
. 2016 Nov;
127(12):3546-3551.
PMID: 27825055
Objective: We sought to understand the alteration in the anisotropic, or direction dependent, character of muscle as measured by electrical impedance myography (EIM) in subjects with Duchenne muscular dystrophy (DMD)...
7.
Govindarajan R, Iyadurai S, Connolly A, Zaidman C
Neuromuscul Disord
. 2015 May;
25(8):651-2.
PMID: 25998611
Neuromuscular junction disorders in children are either genetic, such as congenital myasthenic syndrome, or autoimmune with circulating antibodies most commonly against acetylcholine receptors. There is limited experience recognizing and treating...
8.
Rutkove S, Geisbush T, Mijailovic A, Shklyar I, Pasternak A, Visyak N, et al.
Pediatr Neurol
. 2014 May;
51(1):88-92.
PMID: 24814059
Background: Electrical impedance myography and quantitative ultrasound are two noninvasive, painless, and effort-independent approaches for assessing neuromuscular disease. Both techniques have potential to serve as useful biomarkers in clinical trials...
9.
Cady J, Koval E, Benitez B, Zaidman C, Jockel-Balsarotti J, Allred P, et al.
JAMA Neurol
. 2014 Feb;
71(4):449-53.
PMID: 24535663
Importance: Amyotrophic lateral sclerosis (ALS) is a fatal neurodegenerative disease in which microglia play a significant and active role. Recently, a rare missense variant (p.R47H) in the microglial activating gene...
10.
Harms M, Cady J, Zaidman C, Cooper P, Bali T, Allred P, et al.
Neurobiol Aging
. 2013 Apr;
34(9):2234.e13-9.
PMID: 23597494
Hexanucleotide repeat expansions in C9ORF72 are a common cause of familial and apparently sporadic amyotrophic lateral sclerosis (ALS) and frontal temporal dementia (FTD). The mechanism by which expansions cause neurodegeneration...