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Craig T January

Explore the profile of Craig T January including associated specialties, affiliations and a list of published articles. Areas
Snapshot
Articles 81
Citations 7855
Followers 0
Related Specialties
Cardiology & Vascular Diseases
Physiology
Biochemistry
Top 10 Co-Authors
Brian P Delisle
Corey L Anderson
Timothy J Kamp
L Samuel Wann
Cynthia M Tracy
Michael J Ackerman
Michael D Ezekowitz
Blake D Anson
Lee L Eckhardt
William G Stevenson
Published In
Circulation
Am J Physiol Heart Circ Physiol
J Am Coll Cardiol
Heart Rhythm
Br J Pharmacol
Affiliations
Soon will be listed here.
Recent Articles
11.
2019 AHA/ACC/HRS Focused Update of the 2014 AHA/ACC/HRS Guideline for the Management of Patients With Atrial Fibrillation: A Report of the American College of Cardiology/American Heart Association Task Force on Clinical Practice Guidelines and the...
January C, Samuel Wann L, Calkins H, Chen L, Cigarroa J, Cleveland Jr J, et al.
J Am Coll Cardiol . 2019 Feb; 74(1):104-132. PMID: 30703431
No abstract available.
12.
2019 AHA/ACC/HRS Focused Update of the 2014 AHA/ACC/HRS Guideline for the Management of Patients With Atrial Fibrillation: A Report of the American College of Cardiology/American Heart Association Task Force on Clinical Practice Guidelines and the...
January C, Samuel Wann L, Calkins H, Chen L, Cigarroa J, Cleveland Jr J, et al.
Circulation . 2019 Jan; 140(2):e125-e151. PMID: 30686041
No abstract available.
13.
Atrial Fibrillation and Anticoagulation: One Size Fits All?
Wright J, January C
Ann Intern Med . 2018 Sep; 169(8):575-576. PMID: 30264144
No abstract available.
14.
Visualizing Mutation-Specific Differences in the Trafficking-Deficient Phenotype of Kv11.1 Proteins Linked to Long QT Syndrome Type 2
Hall A, Anderson C, Smith J, Mirshahi T, Elayi C, January C, et al.
Front Physiol . 2018 Jun; 9:584. PMID: 29875689
encodes the Kv11.1 α-subunit that underlies the rapidly activating delayed-rectifier K current in the heart. Loss-of-function mutations cause long QT syndrome type 2 (LQT2), and most LQT2-linked missense mutations inhibit...
15.
Functional Invalidation of Putative Sudden Infant Death Syndrome-Associated Variants in the -Encoded Kv11.1 Channel
Smith J, Tester D, Hall A, Burgess D, Hsu C, Elayi S, et al.
Circ Arrhythm Electrophysiol . 2018 May; 11(5):e005859. PMID: 29752375
Background: Heterologous functional validation studies of putative long-QT syndrome subtype 2-associated variants clarify their pathological potential and identify disease mechanism(s) for most variants studied. The purpose of this study is...
16.
Advancing precision medicine for the treatment of long-QT syndrome type 2: shedding light on lumacaftor
Delisle B, January C
Eur Heart J . 2017 Oct; 39(16):1456-1458. PMID: 29040441
No abstract available.
17.
Flecainide treats a novel mutation associated with Andersen-Tawil syndrome
Van Ert H, McCune E, Orland K, Maginot K, Von Bergen N, January C, et al.
HeartRhythm Case Rep . 2017 May; 3(2):151-154. PMID: 28491792
No abstract available.
18.
Molecular pathogenesis of long QT syndrome type 2
Smith J, Anderson C, Burgess D, Elayi C, January C, Delisle B
J Arrhythm . 2016 Oct; 32(5):373-380. PMID: 27761161
The molecular mechanisms underlying congenital long QT syndrome (LQTS) are now beginning to be understood. New insights into the etiology and therapeutic strategies are emerging from heterologous expression studies of...
19.
Identification of Drug-Drug Interactions In Vitro: A Case Study Evaluating the Effects of Sofosbuvir and Amiodarone on hiPSC-Derived Cardiomyocytes
Millard D, Strock C, Carlson C, Aoyama N, Juhasz K, Goetze T, et al.
Toxicol Sci . 2016 Aug; 154(1):174-182. PMID: 27503387
Drug-drug interactions pose a difficult drug safety problem, given the increasing number of individuals taking multiple medications and the relative complexity of assessing the potential for interactions. For example, sofosbuvir-based...
20.
IK1-enhanced human-induced pluripotent stem cell-derived cardiomyocytes: an improved cardiomyocyte model to investigate inherited arrhythmia syndromes
Vaidyanathan R, Markandeya Y, Kamp T, Makielski J, January C, Eckhardt L
Am J Physiol Heart Circ Physiol . 2016 Apr; 310(11):H1611-21. PMID: 27059077
Currently available induced pluripotent stem cell-derived cardiomyocytes (iPS-CMs) do not ideally model cellular mechanisms of human arrhythmic disease due to lack of a mature action potential (AP) phenotype. In this...