Corey L Anderson
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Explore the profile of Corey L Anderson including associated specialties, affiliations and a list of published articles.
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24
Citations
716
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Recent Articles
1.
Reilly L, Josvai M, Kalluri M, Anderson C, Ni H, Orland K, et al.
JACC Clin Electrophysiol
. 2024 Sep;
10(12):2628-2632.
PMID: 39297840
Idiopathic ventricular fibrillation (IVF) is an unrefined diagnosis representing a heterogeneous patient group without a structural or genetic definition. IVF treatment is not mechanistic-based due to the lack of experimental...
2.
Anderson C, Brown K, North R, Walters J, Kaska S, Wolff M, et al.
J Proteome Res
. 2024 May;
23(6):1970-1982.
PMID: 38718259
Lamin A/C (LMNA) is an important component of nuclear lamina. Mutations cause arrhythmia, heart failure, and sudden cardiac death. While LMNA-associated cardiomyopathy typically has an aggressive course that responds poorly...
3.
Anderson C, Munawar S, Reilly L, Kamp T, January C, Delisle B, et al.
Front Cardiovasc Med
. 2022 Jul;
9:900431.
PMID: 35859585
Over the last two decades, an exponentially expanding number of genetic variants have been identified associated with inherited cardiac conditions. These tremendous gains also present challenges in deciphering the clinical...
4.
Kekenes-Huskey P, Burgess D, Sun B, Bartos D, Rozmus E, Anderson C, et al.
Int J Mol Sci
. 2022 Jul;
23(13).
PMID: 35806392
The electrocardiogram (ECG) empowered clinician scientists to measure the electrical activity of the heart noninvasively to identify arrhythmias and heart disease. Shortly after the standardization of the 12-lead ECG for...
5.
Anderson C, Langer E, Routes T, McWilliams S, Bereslavskyy I, Kamp T, et al.
NPJ Genom Med
. 2021 Dec;
6(1):103.
PMID: 34862408
Hundreds of LMNA variants have been associated with several distinct disease phenotypes. However, genotype-phenotype relationships remain largely undefined and the impact for most variants remains unknown. We performed a functional...
6.
Feng L, Zhang J, Lee C, Kim G, Liu F, Petersen A, et al.
Circ Arrhythm Electrophysiol
. 2021 Mar;
14(4):e009343.
PMID: 33729832
[Figure: see text].
7.
Ono M, Burgess D, Schroder E, Elayi C, Anderson C, January C, et al.
Biomolecules
. 2020 Aug;
10(8).
PMID: 32759882
Significant advances in our understanding of the molecular mechanisms that cause congenital long QT syndrome (LQTS) have been made. A wide variety of experimental approaches, including heterologous expression of mutant...
8.
Anderson C, Routes T, Eckhardt L, Delisle B, January C, Kamp T
Genet Med
. 2020 Jun;
22(10):1642-1652.
PMID: 32475984
Purpose: DNA sequencing technology has unmasked a vast number of uncharacterized single-nucleotide variants in disease-associated genes, and efficient methods are needed to determine pathogenicity and enable clinical care. Methods: We...
9.
Hall A, Anderson C, Smith J, Mirshahi T, Elayi C, January C, et al.
Front Physiol
. 2018 Jun;
9:584.
PMID: 29875689
encodes the Kv11.1 α-subunit that underlies the rapidly activating delayed-rectifier K current in the heart. Loss-of-function mutations cause long QT syndrome type 2 (LQT2), and most LQT2-linked missense mutations inhibit...
10.
Smith J, Tester D, Hall A, Burgess D, Hsu C, Elayi S, et al.
Circ Arrhythm Electrophysiol
. 2018 May;
11(5):e005859.
PMID: 29752375
Background: Heterologous functional validation studies of putative long-QT syndrome subtype 2-associated variants clarify their pathological potential and identify disease mechanism(s) for most variants studied. The purpose of this study is...