Coeli M B Lopes
Overview
Explore the profile of Coeli M B Lopes including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
1353
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Recent Articles
1.
Braun C, Parks X, Qudsi H, Lopes C
Commun Biol
. 2021 Dec;
4(1):1392.
PMID: 34907346
Plasma membrane phosphatidylinositol 4-phosphate (PI4P) is a precursor of PI(4,5)P, an important regulator of a large number of ion channels. Although the role of the phospholipid PI(4,5)P in stabilizing ion...
2.
Parks X, Qudsi H, Braun C, Lopes C
PLoS One
. 2020 Aug;
15(8):e0237591.
PMID: 32833978
The slow cardiac delayed rectifier current (IKs) is formed by KCNQ1 and KCNE1 subunits and is one of the major repolarizing currents in the heart. Decrease of IKs currents either...
3.
O-Uchi J, Sorenson J, Jhun B, Mishra J, Hurst S, Williams K, et al.
Biochem Biophys Res Commun
. 2015 Aug;
465(3):464-70.
PMID: 26277396
Protein kinase C (PKC) plays key roles in the regulation of signal transduction and cellular function in various cell types. At least ten PKC isoforms have been identified and intracellular...
4.
Jhun B, O-Uchi J, Wang W, Ha C, Zhao J, Kim J, et al.
Circ Res
. 2011 Nov;
110(1):59-70.
PMID: 22076634
Rationale: The Rad-Gem/Kir-related family (RGKs) consists of small GTP-binding proteins that strongly inhibit the activity of voltage-gated calcium channels. Among RGKs, Rem1 is strongly and specifically expressed in cardiac tissue....
5.
Jons C, O-Uchi J, Moss A, Reumann M, Rice J, Goldenberg I, et al.
Sci Transl Med
. 2011 Apr;
3(76):76ra28.
PMID: 21451124
Inherited long QT syndrome (LQTS) is caused by mutations in ion channels that delay cardiac repolarization, increasing the risk of sudden death from ventricular arrhythmias. Currently, the risk of sudden...
6.
Williams D, Lopes C, Rosenhouse-Dantsker A, Connelly H, Matavel A, O-Uchi J, et al.
J Am Soc Nephrol
. 2010 Nov;
21(12):2117-29.
PMID: 21088294
SeSAME/EAST syndrome is a channelopathy consisting of a hypokalemic, hypomagnesemic, metabolic alkalosis associated with seizures, sensorineural deafness, ataxia, and developmental abnormalities. This disease links to autosomal recessive mutations in KCNJ10,...
7.
8.
Horr S, Goldenberg I, Moss A, O-Uchi J, Barsheshet A, Connelly H, et al.
J Cardiovasc Electrophysiol
. 2010 Jul;
22(2):193-200.
PMID: 20662986
Unlabelled: Background: Data regarding possible ion channel mechanisms that predispose to ventricular tachyarrhythmias in patients with phenotype-negative long-QT syndrome (LQTS) are limited. Methods And Results: We carried out cellular expression...
9.
Casey L, Pistner A, Belmonte S, Migdalovich D, Stolpnik O, Nwakanma F, et al.
Circ Res
. 2010 Jun;
107(4):532-9.
PMID: 20576935
Rationale: Excess signaling through cardiac Gbetagamma subunits is an important component of heart failure (HF) pathophysiology. They recruit elevated levels of cytosolic G protein-coupled receptor kinase (GRK)2 to agonist-stimulated beta-adrenergic...
10.
Wojtovich A, Williams D, Karcz M, Lopes C, Gray D, Nehrke K, et al.
Circ Res
. 2010 Feb;
106(7):1190-6.
PMID: 20185796
Rationale: The mitochondrial ATP sensitive potassium channel (mK(ATP)) is implicated in cardioprotection by ischemic preconditioning (IPC), but the molecular identity of the channel remains controversial. The validity of current methods...