Cindy L Vnencak-Jones
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Explore the profile of Cindy L Vnencak-Jones including associated specialties, affiliations and a list of published articles.
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54
Citations
1981
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Recent Articles
21.
Meador C, Micheel C, Levy M, Lovly C, Horn L, Warner J, et al.
Clin Cancer Res
. 2014 Mar;
20(9):2264-75.
PMID: 24599935
Increased understanding of intertumoral heterogeneity at the genomic level has led to significant advancements in the treatment of solid tumors. Functional genomic alterations conferring sensitivity to targeted therapies can take...
22.
Hutchinson K, Lipson D, Stephens P, Otto G, Lehmann B, Lyle P, et al.
Clin Cancer Res
. 2013 Dec;
19(24):6696-702.
PMID: 24345920
Purpose: Recurrent "driver" mutations at specific loci in BRAF, NRAS, KIT, GNAQ, and GNA11 define clinically relevant molecular subsets of melanoma, but more than 30% are "pan-negative" for these recurrent...
23.
Jones H, Gold M, Giannico G, Troutman A, Vnencak-Jones C, Schultenover S, et al.
Int J Gynecol Pathol
. 2013 Dec;
33(1):64-73.
PMID: 24300538
This report describes the clinicopathologic features of a primary lymphoepithelioma-like carcinoma of the endometrium, representing only the fourth reported case of this tumor at this location. In addition to its...
24.
Alfaro M, Cohen M, Vnencak-Jones C
Am J Med Genet A
. 2013 Aug;
161A(10):2620-5.
PMID: 23949867
Fragile X syndrome results from an expansion of the CGG trinucleotide repeat in the 5' untranslated region of the Fragile X Mental Retardation 1 (FMR1) gene. Expansion of a maternal...
25.
Cushman-Vokoun A, Stover D, Zhao Z, Koehler E, Berlin J, Vnencak-Jones C
Clin Colorectal Cancer
. 2013 Jun;
12(3):168-78.
PMID: 23773459
Background: Molecular analysis has become important in colorectal carcinoma (CRC) evaluation. Alterations in KRAS, BRAF, or mismatch repair (MMR) genes may determine therapeutic response or define a hereditary cancer syndrome....
26.
Ohashi K, Sequist L, Arcila M, Lovly C, Chen X, Rudin C, et al.
Clin Cancer Res
. 2013 Mar;
19(9):2584-91.
PMID: 23515407
Purpose: We sought to determine the frequency and clinical characteristics of patients with lung cancer harboring NRAS mutations. We used preclinical models to identify targeted therapies likely to be of...
27.
Dahlman K, Xia J, Hutchinson K, Ng C, Hucks D, Jia P, et al.
Cancer Discov
. 2012 Jul;
2(9):791-7.
PMID: 22798288
Unlabelled: Kinase inhibitors are accepted treatment for metastatic melanomas that harbor specific driver mutations in BRAF or KIT, but only 40% to 50% of cases are positive. To uncover other...
28.
Ohashi K, Sequist L, Arcila M, Moran T, Chmielecki J, Lin Y, et al.
Proc Natl Acad Sci U S A
. 2012 Jul;
109(31):E2127-33.
PMID: 22773810
Acquired resistance to EGF receptor (EGFR) tyrosine kinase inhibitors (TKIs) is inevitable in metastatic EGFR-mutant lung cancers. Here, we modeled disease progression using EGFR-mutant human tumor cell lines. Although five...
29.
Lovly C, Dahlman K, Fohn L, Su Z, Dias-Santagata D, Hicks D, et al.
PLoS One
. 2012 Apr;
7(4):e35309.
PMID: 22536370
Purpose: Knowledge of tumor mutation status is becoming increasingly important for the treatment of cancer, as mutation-specific inhibitors are being developed for clinical use that target only sub-populations of patients...
30.
Goins W, Edwards K, Vnencak-Jones C, Rock M, Swift M, Thayer V, et al.
Clin Infect Dis
. 2012 Jan;
54(7):938-45.
PMID: 22238169
Background: Antibiotic postexposure prophylaxis (PEP) following pertussis exposure is recommended but has never been evaluated in healthcare personnel (HCP) vaccinated with acellular pertussis vaccine (Tdap). Methods: Tdap-vaccinated HCP were randomized...