» Authors » Catherine D Falkenstein

Catherine D Falkenstein

Explore the profile of Catherine D Falkenstein including associated specialties, affiliations and a list of published articles. Areas
Snapshot
Articles 5
Citations 15
Followers 0
Related Specialties
Top 10 Co-Authors
Published In
Affiliations
Soon will be listed here.
Recent Articles
1.
Ding Y, Sussman J, Madden K, Loftus J, Chen R, Falkenstein C, et al.
Blood . 2025 Jan; 145(11):1195-1210. PMID: 39774844
Philadelphia chromosome-like B-cell acute lymphoblastic leukemia (Ph-like ALL) is driven by genetic alterations that induce constitutive kinase signaling and is associated with chemoresistance and high relapse risk in children and...
2.
Torres-Diz M, Reglero C, Falkenstein C, Castro A, Hayer K, Radens C, et al.
Cancer Res . 2024 Aug; 84(20):3327-3336. PMID: 39094066
Relapsed or refractory B-cell acute lymphoblastic leukemia (B-ALL) is a major cause of pediatric cancer-related deaths. Relapse-specific mutations do not account for all chemotherapy failures in B-ALL patients, suggesting additional...
3.
Torres-Diz M, Reglero C, Falkenstein C, Castro A, Hayer K, Radens C, et al.
bioRxiv . 2024 Aug; PMID: 39091882
Relapsed or refractory B-cell acute lymphoblastic leukemia (B-ALL) is a major cause of pediatric cancer-related deaths. Relapse-specific mutations do not account for all chemotherapy failures in B- ALL patients, suggesting...
4.
Ang Z, Paruzzo L, Hayer K, Schmidt C, Torres Diz M, Xu F, et al.
Blood . 2023 Sep; 142(20):1724-1739. PMID: 37683180
Aberrant skipping of coding exons in CD19 and CD22 compromises the response to immunotherapy in B-cell malignancies. Here, we showed that the MS4A1 gene encoding human CD20 also produces several...
5.
Ang Z, Paruzzo L, Hayer K, Schmidt C, Torres Diz M, Xu F, et al.
bioRxiv . 2023 Aug; PMID: 37645778
Key Points: In normal & malignant human B cells, CD20 mRNA is alternatively spliced into four 5'-UTR isoforms, some of which are translation-deficient.The balance between translation-deficient and -competent isoforms modulates...