Caitlin S Latimer
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Explore the profile of Caitlin S Latimer including associated specialties, affiliations and a list of published articles.
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979
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Recent Articles
1.
Samstag C, Chapman N, Gibbons L, Geller J, Loeb N, Dharap S, et al.
Alzheimers Dement
. 2024 Dec;
21(2):e14428.
PMID: 39713867
Introduction: We investigated whether the cerebellum develops neuropathology that correlates with well-accepted Alzheimer's disease (AD) neuropathological markers and cognitive status. Methods: We studied cerebellar cytoarchitecture in a cohort (N = ...
2.
Gabitto M, Travaglini K, Rachleff V, Kaplan E, Long B, Ariza J, et al.
Nat Neurosci
. 2024 Oct;
27(12):2366-2383.
PMID: 39402379
Alzheimer's disease (AD) is the leading cause of dementia in older adults. Although AD progression is characterized by stereotyped accumulation of proteinopathies, the affected cellular populations remain understudied. Here we...
3.
Scherpelz K, Yoda R, Jayadev S, Davis M, Hincks J, Liachko N, et al.
Neuropathology
. 2024 Oct;
PMID: 39391989
Hereditary spastic paraplegia (HSP) with thin corpus callosum can be due to a variety of genetic causes, the most common of which are biallelic variants in SPG11 (HSP11). Only six...
4.
Froines C, Lin A, Pakzad-Vaezi K, Juric-Sekhar G, Latimer C, Scherpelz K, et al.
GMS Ophthalmol Cases
. 2024 Oct;
14:Doc13.
PMID: 39385769
Purpose: Highlight an unusual case of epithelial downgrowth (EDG) masquerading as granulomatous anterior and intermediate uveitis with histopathologic evidence of 5-fluorouracil (5-FU) treatment. Case Description: A 33-year-old man presented after...
5.
Braun M, Simon M, Jang J, Sanderson K, Swierz J, Sevao M, et al.
bioRxiv
. 2024 Sep;
PMID: 39229234
The appearance of misfolded and aggregated proteins is a pathological hallmark of numerous neurodegenerative diseases including Alzheimer's disease and Parkinson's disease. Sleep disruption is proposed to contribute to these pathological...
6.
Domoto-Reilly K, Distad B, Miller D, Lin Y, Ivanick D, Warren A, et al.
Neurol Genet
. 2024 Jul;
10(4):e200173.
PMID: 39055961
Objectives: Here, we report detailed clinicopathologic evaluation of 2 individuals with pathogenic variants in , including one novel likely pathogenic splice variant. We describe the striking diversity of clinical phenotypes...
7.
Gazula H, Tregidgo H, Billot B, Balbastre Y, Williams-Ramirez J, Herisse R, et al.
Elife
. 2024 Jun;
12.
PMID: 38896568
We present open-source tools for three-dimensional (3D) analysis of photographs of dissected slices of human brains, which are routinely acquired in brain banks but seldom used for quantitative analysis. Our...
8.
Barsoum S, Latimer C, Nolan A, Barrett A, Chang K, Troncoso J, et al.
bioRxiv
. 2024 May;
PMID: 38766087
Despite the presence of significant Alzheimer's disease (AD) pathology, characterized by amyloid β (Aβ) plaques and phosphorylated tau (pTau) tangles, some cognitively normal elderly individuals do not inevitably develop dementia....
9.
Jadhav V, Stair J, Eck R, Smukowski S, Currey H, Toscano L, et al.
Neurobiol Dis
. 2024 Feb;
193:106441.
PMID: 38378122
Alzheimer's disease (AD), the most common aging-associated neurodegenerative dementia disorder, is defined by the presence of amyloid beta (Aβ) and tau aggregates in the brain. However, more than half of...
10.
Cholerton B, Latimer C, Crane P, Corrada M, Gibbons L, Larson E, et al.
Neurology
. 2024 Jan;
102(3):e208060.
PMID: 38175995
Background And Objectives: The aim of this study was to compare 2 large clinicopathologic cohorts of participants aged 90+ and to determine whether the association between neuropathologic burden and dementia...