Suman Jayadev
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Explore the profile of Suman Jayadev including associated specialties, affiliations and a list of published articles.
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87
Citations
1577
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Recent Articles
1.
Bateman R, Li Y, McDade E, Llibre-Guerra J, Clifford D, Atri A, et al.
medRxiv
. 2025 Feb;
PMID: 39974075
Background: Amyloid-plaque removal by monoclonal antibody therapies slows clinical progression in symptomatic Alzheimer's disease; however, the potential for delaying the onset of clinical symptoms in asymptomatic people is unknown. The...
2.
Karagas N, Young J, Blue E, Jayadev S
Neurol Genet
. 2025 Jan;
11(1):e200224.
PMID: 39885961
Alzheimer disease (AD), the most common dementing syndrome in the United States, is currently established by the presence of amyloid-β and tau protein biomarkers in the setting of clinical cognitive...
3.
Reid A, Jayadev S, Prater K
Glia
. 2025 Jan;
73(3):519-538.
PMID: 39760224
Human genetics studies lent firm evidence that microglia are key to Alzheimer's disease (AD) pathogenesis over a decade ago following the identification of AD-associated genes that are expressed in a...
4.
Weinstein J, Jayadev S, Liddelow S, Eggen B
Glia
. 2024 Nov;
73(3):448-450.
PMID: 39587765
No abstract available.
5.
Knox K, Davidson S, Lehmann L, Skinner E, Lo A, Jayadev S, et al.
bioRxiv
. 2024 Oct;
PMID: 39416203
Introduction: Alzheimer's disease (AD) patients are at greater risk of focal seizures than similarly aged adults; these seizures, left untreated, may worsen functional decline. Older people with epilepsy generally respond...
6.
Gabitto M, Travaglini K, Rachleff V, Kaplan E, Long B, Ariza J, et al.
Nat Neurosci
. 2024 Oct;
27(12):2366-2383.
PMID: 39402379
Alzheimer's disease (AD) is the leading cause of dementia in older adults. Although AD progression is characterized by stereotyped accumulation of proteinopathies, the affected cellular populations remain understudied. Here we...
7.
Scherpelz K, Yoda R, Jayadev S, Davis M, Hincks J, Liachko N, et al.
Neuropathology
. 2024 Oct;
PMID: 39391989
Hereditary spastic paraplegia (HSP) with thin corpus callosum can be due to a variety of genetic causes, the most common of which are biallelic variants in SPG11 (HSP11). Only six...
8.
Domoto-Reilly K, Distad B, Miller D, Lin Y, Ivanick D, Warren A, et al.
Neurol Genet
. 2024 Jul;
10(4):e200173.
PMID: 39055961
Objectives: Here, we report detailed clinicopathologic evaluation of 2 individuals with pathogenic variants in , including one novel likely pathogenic splice variant. We describe the striking diversity of clinical phenotypes...
9.
Mishra S, Morshed N, Sindhu S, Kinoshita C, Stevens B, Jayadev S, et al.
bioRxiv
. 2024 Jul;
PMID: 38979155
The gene encodes the sortilin related receptor protein SORLA, a sorting receptor that regulates endo-lysosomal trafficking of various substrates. Loss of function variants in are causative for Alzheimer's disease (AD)...
10.
Thomas P, Leclerc M, Evitts K, Brown C, Miller W, Hanson A, et al.
Alzheimers Dement (Amst)
. 2024 May;
16(2):e12603.
PMID: 38800123
Introduction: Brain insulin resistance and deficiency is a consistent feature of Alzheimer's disease (AD). Insulin resistance can be mediated by the surface expression of the insulin receptor (IR). Cleavage of...