C Maasch
Overview
Explore the profile of C Maasch including associated specialties, affiliations and a list of published articles.
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Articles
14
Citations
296
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0
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Recent Articles
1.
Hoehlig K, Johnson K, Pryazhnikov E, Maasch C, Clemens-Smith A, Purschke W, et al.
Br J Pharmacol
. 2015 Feb;
172(12):3086-98.
PMID: 25659966
Background And Purpose: Calcitonin gene-related peptide (CGRP) plays an important role in the pathology of migraine, and recent clinical trials suggest the inhibition of CGRP-mediated processes as a new therapeutic...
2.
Vater A, Sahlmann J, Kroger N, Zollner S, Lioznov M, Maasch C, et al.
Clin Pharmacol Ther
. 2013 Apr;
94(1):150-7.
PMID: 23588307
NOX-A12 is a PEGylated mirror-image oligonucleotide (a so-called Spiegelmer) that binds to CXCL12 (stromal cell-derived factor-1, SDF-1) with high affinity thereby inhibiting CXCL12 signaling on both its receptors, CXCR4 and...
3.
Maasch C, Wagner S, Lindschau C, Alexander G, Buchner K, Gollasch M, et al.
FASEB J
. 2000 Aug;
14(11):1653-63.
PMID: 10929000
Protein kinase C (PKC) isoforms exert specific intracellular functions, but the different isoforms display little substrate specificity in vitro. Selective PKC isoform targeting may be a mechanism to achieve specificity....
4.
Kang N, Alexander G, Park J, Maasch C, Buchwalow I, Luft F, et al.
Kidney Int
. 1999 Nov;
56(5):1737-50.
PMID: 10571782
Background: The cellular effects of hyperglycemia are mediated by protein kinase C (PKC). However, PKC consists of several distinct isoforms, and their contribution to the pathogenesis of diabetic complications in...
5.
Li P, Maasch C, Haller H, Dietz R, von Harsdorf R
Circulation
. 1999 Sep;
100(9):967-73.
PMID: 10468528
Background: Vascular smooth muscle cell (VSMC) apoptosis is a component of a variety of cardiovascular diseases and may be related to reactive oxygen species (ROS). This study was designed to...
6.
Hempel A, Lindschau C, Maasch C, Mahn M, Bychkov R, Noll T, et al.
Circulation
. 1999 May;
99(19):2523-9.
PMID: 10330383
Background: Dihydropyridines block calcium channels; however, they also influence endothelial cells, which do not express calcium channels. We tested the hypothesis that nifedipine can prevent ischemia-induced endothelial permeability increases by...
7.
Wellner M, Maasch C, Kupprion C, Lindschau C, Luft F, Haller H
Arterioscler Thromb Vasc Biol
. 1999 Jan;
19(1):178-85.
PMID: 9888881
The heparin-binding protein vascular endothelial growth factor (VEGF) is a highly specific growth factor for endothelial cells. VEGF binds to specific tyrosine kinase receptors, which mediate intracellular signaling. We investigated...
8.
Haller H, Maasch C, Lindschau C, Brachmann M, Buchner K, Luft F
Acta Physiol Scand
. 1999 Jan;
164(4):599-609.
PMID: 9887982
Protein kinase C is an important second messenger system, which is translocated from the cytosol to the cell membrane upon cell stimulation. We used confocal microscopy to study the spatial...
9.
Haller H, Hempel A, Homuth V, Mandelkow A, Busjahn A, Maasch C, et al.
Lancet
. 1998 Sep;
351(9107):945-9.
PMID: 9734941
Background: Oedema and vascular leakage play a part in the pathogenesis of pre-eclampsia. We tested the hypothesis that serum from pre-eclamptic patients increases endothelial-cell permeability and examined possible signal-transduction pathways....
10.
Dragun D, Tullius S, Park J, Maasch C, Lukitsch I, Lippoldt A, et al.
Kidney Int
. 1998 Aug;
54(2):590-602.
PMID: 9690227
Background: Ischemia-reperfusion injury after organ transplantation is a major cause of delayed graft function. We showed earlier that antisense oligodesoxynucleotides (ODN) for intercellular adhesion molecule-1 (ICAM-1) ameliorate reperfusion injury after...