C K Weber
Overview
Explore the profile of C K Weber including associated specialties, affiliations and a list of published articles.
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21
Citations
595
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Recent Articles
1.
Grossmayer G, Munoz L, Weber C, Franz S, Voll R, Kern P, et al.
Ann Rheum Dis
. 2008 Jan;
67(11):1626-32.
PMID: 18165321
Objective: Accumulation of dying and dead cells is thought to be involved in the etiopathogenesis of systemic lupus erythaematosus (SLE). Clearance has been described mainly for apoptotic cells; however, the...
2.
Weber C, Adler G
Curr Opin Gastroenterol
. 2006 Oct;
17(5):426-9.
PMID: 17031195
The pathophysiologic concept of acute pancreatitis focuses on early events inside acinar cells such as activation of trypsin. Despite significant progress in the understanding of molecular events in experimental pancreatitis,...
3.
Aschoff A, Juchems M, Weber C, Brambs H
Z Gastroenterol
. 2004 Oct;
42(10):1199-205.
PMID: 15508062
This article reviews published data regarding CT colonography and discusses both technical and medical aspects of its development over the last 10 years. Although colonography can be performed using MRI...
4.
Greten F, Wagner M, Weber C, Zechner U, Adler G, Schmid R
Pancreatology
. 2002 Jul;
1(4):363-8.
PMID: 12120215
Pancreatic cancer is a devastating disease with a fatal prognosis due to late diagnosis and resistance to radiation and chemotherapy. The average survival after diagnosis is still 3 to 8...
5.
Weber C, Adler G
Pancreatology
. 2002 Jul;
1(4):356-62.
PMID: 12120214
Acute pancreatitis represents a local inflammatory disorder with severe systemic consequences. Significant progress in understanding the pathophysiology of acute pancreatitis has been achieved in recent years. However, there is no...
6.
Biglycan is overexpressed in pancreatic cancer and induces G1-arrest in pancreatic cancer cell lines
Weber C, Sommer G, Michl P, Fensterer H, Weimer M, Gansauge F, et al.
Gastroenterology
. 2001 Aug;
121(3):657-67.
PMID: 11522750
Background & Aims: Biglycan (PG-I), a component of the extracellular matrix (ECM), is overexpressed in pancreatic cancer. To determine possible matrix-tumor interactions, we investigated the effects of PG-I on pancreatic...
7.
Weber C, Slupsky J, Kalmes H, Rapp U
Cancer Res
. 2001 Apr;
61(9):3595-8.
PMID: 11325826
Growth factor-induced signalling leads to activation of members of the Ras family and subsequent stimulation of different Raf isoforms. Within the mechanism of Raf activation, two isoforms of Raf, cRaf...
8.
Wagner M, Greten F, Weber C, Koschnick S, Mattfeldt T, Deppert W, et al.
Genes Dev
. 2001 Feb;
15(3):286-93.
PMID: 11159909
This study describes a tumor progression model for ductal pancreatic cancer in mice overexpressing TGF-alpha. Activation of Ras and Erk causes induction of cyclin D1-Cdk4 without increase of cyclin E...
9.
Weber C, Liptay S, Wirth T, Adler G, Schmid R
Gastroenterology
. 2000 Oct;
119(5):1209-18.
PMID: 11054378
Background & Aims: Activation of NF-kappaB/Rel has been implicated in the pathogenesis of inflammatory bowel disease (IBD). Various drugs used in the treatment of IBD, such as glucocorticoids, 5-aminosalicylic acid,...
10.
Baumann B, Weber C, Troppmair J, Whiteside S, Israel A, Rapp U, et al.
Proc Natl Acad Sci U S A
. 2000 Apr;
97(9):4615-20.
PMID: 10758165
NF-kappaB is regulated by inhibitor proteins (IkappaBs), which retain NF-kappaB in the cytoplasm. Signal-induced phosphorylation by the IkappaB-kinase complex containing the IkappaB-kinases 1 and 2 (IKK-1/2 or IKK-alpha/beta) and subsequent...