Brittany C Michel
Overview
Explore the profile of Brittany C Michel including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
10
Citations
884
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Otto J, Ursu O, Wu A, Winter E, Cuoco M, Ma S, et al.
Mol Cell
. 2023 Apr;
83(8):1350-1367.e7.
PMID: 37028419
The mammalian SWI/SNF (mSWI/SNF or BAF) family of chromatin remodeling complexes play critical roles in regulating DNA accessibility and gene expression. The three final-form subcomplexes-cBAF, PBAF, and ncBAF-are distinct in...
2.
Michel B, Fulp S, Drayton D, White K
J STEM Outreach
. 2022 Mar;
4(3).
PMID: 35252782
STEM training of college-bound and college students has reliably employed hands-on experiential learning by placing students in on-campus research settings. Dana-Farber/Harvard Cancer Center's Young Empowered Scientists for ContinUed Research Engagement...
3.
Mashtalir N, Dao H, Sankar A, Liu H, Corin A, Bagert J, et al.
Science
. 2021 Aug;
373(6552):306-315.
PMID: 34437148
Mammalian SWI/SNF (mSWI/SNF) adenosine triphosphate-dependent chromatin remodelers modulate genomic architecture and gene expression and are frequently mutated in disease. However, the specific chromatin features that govern their nucleosome binding and...
4.
Barish S, Barakat T, Michel B, Mashtalir N, Phillips J, Valencia A, et al.
Am J Hum Genet
. 2020 Nov;
107(6):1096-1112.
PMID: 33232675
SWI/SNF-related intellectual disability disorders (SSRIDDs) are rare neurodevelopmental disorders characterized by developmental disability, coarse facial features, and fifth digit/nail hypoplasia that are caused by pathogenic variants in genes that encode...
5.
Inoue D, Chew G, Liu B, Michel B, Pangallo J, DAvino A, et al.
Nature
. 2019 Oct;
574(7778):432-436.
PMID: 31597964
SF3B1 is the most commonly mutated RNA splicing factor in cancer, but the mechanisms by which SF3B1 mutations promote malignancy are poorly understood. Here we integrated pan-cancer splicing analyses with...
6.
Michel B, DAvino A, Cassel S, Mashtalir N, McKenzie Z, McBride M, et al.
Nat Cell Biol
. 2018 Nov;
20(12):1410-1420.
PMID: 30397315
Mammalian SWI/SNF chromatin remodelling complexes exist in three distinct, final-form assemblies: canonical BAF (cBAF), PBAF and a newly characterized non-canonical complex (ncBAF). However, their complex-specific targeting on chromatin, functions and...
7.
Mashtalir N, DAvino A, Michel B, Luo J, Pan J, Otto J, et al.
Cell
. 2018 Oct;
175(5):1272-1288.e20.
PMID: 30343899
Mammalian SWI/SNF (mSWI/SNF) ATP-dependent chromatin remodeling complexes are multi-subunit molecular machines that play vital roles in regulating genomic architecture and are frequently disrupted in human cancer and developmental disorders. To...
8.
Pan J, Meyers R, Michel B, Mashtalir N, Sizemore A, Wells J, et al.
Cell Syst
. 2018 May;
6(5):555-568.e7.
PMID: 29778836
Protein complexes are assemblies of subunits that have co-evolved to execute one or many coordinated functions in the cellular environment. Functional annotation of mammalian protein complexes is critical to understanding...
9.
Michel B, Kadoch C
Nat Genet
. 2017 Apr;
49(5):655-657.
PMID: 28442792
Mammalian SWI/SNF complexes have critical roles in development and differentiation, and are implicated in the pathogenesis of several diseases; however, the mechanisms underpinning disease manifestation and the specificity of the...
10.
Derman A, Nonejuie P, Michel B, Truong B, Fujioka A, Erb M, et al.
J Bacteriol
. 2012 Mar;
194(10):2715-24.
PMID: 22427628
Alp7A is a bacterial actin from Bacillus subtilis plasmid pLS20 that functions in plasmid segregation. Alp7A's function requires that it assemble into filaments that treadmill and exhibit dynamic instability. These...