Bertrand Friguet
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Explore the profile of Bertrand Friguet including associated specialties, affiliations and a list of published articles.
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124
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3449
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Recent Articles
1.
Nina-Diogo A, Hyzewicz J, Hamon M, Forte J, Thorimbert S, Friguet B, et al.
Chembiochem
. 2024 May;
25(19):e202400093.
PMID: 38695553
Oxidative stress is a cellular disorder implicated in various severe diseases and redox biology and represents an important field of research for the last decades. One of the major consequences...
2.
Tighanimine K, Nabuco Leva Ferreira Freitas J, Nemazanyy I, Bankole A, Benarroch-Popivker D, Brodesser S, et al.
Nat Metab
. 2024 Feb;
6(2):323-342.
PMID: 38409325
Cellular senescence affects many physiological and pathological processes and is characterized by durable cell cycle arrest, an inflammatory secretory phenotype and metabolic reprogramming. Here, by using dynamic transcriptome and metabolome...
3.
Medali T, Couchie D, Mougenot N, Mihoc M, Bergmann O, Derks W, et al.
FASEB J
. 2023 Dec;
38(1):e23291.
PMID: 38095283
Myocardial infarction (MI) is characterized by a significant loss of cardiomyocytes (CMs), and it is suggested that reactive oxygen species (ROS) are involved in cell cycle arrest, leading to impaired...
4.
Halkoum R, Salnot V, Capallere C, Plaza C, Lhonore A, Pays K, et al.
J Invest Dermatol
. 2021 Dec;
142(8):2068-2078.e7.
PMID: 34971698
Senescence is a well-characterized cellular state associated with specific markers such as permanent cell proliferation arrest and the secretion of messenger molecules by cells expressing the senescence-associated secretory phenotype. The...
5.
Frasca D, Saada Y, Garcia D, Friguet B
Mech Ageing Dev
. 2020 Dec;
194:111428.
PMID: 33383073
Many cellular stresses induce cellular senescence and the irreversible arrest of cell proliferation in different cell types. Although blocked in their capacity to divide, senescent cells are metabolically active and...
6.
Cohen A, Kennedy B, Anglas U, Bronikowski A, Deelen J, Dufour F, et al.
Mech Ageing Dev
. 2020 Jul;
191:111316.
PMID: 32693105
At a recent symposium on aging biology, a debate was held as to whether or not we know what biological aging is. Most of the participants were struck not only...
7.
Hamon M, Gergondey R, Lhonore A, Friguet B
Free Radic Biol Med
. 2020 Jan;
148:83-95.
PMID: 31904544
The ATP-dependent Lon protease is located in the mitochondrial matrix and oxidized proteins are among its primary targets for their degradation. Impairment of mitochondrial morphology and function together with apoptosis...
8.
Hamon M, Ahmed E, Baraibar M, Friguet B
Proteomics
. 2019 Sep;
20(5-6):e1800421.
PMID: 31507063
Accumulation of oxidatively modified proteins is a hallmark of organismal aging in vivo and of cellular replicative senescence in vitro. Failure of protein maintenance is a major contributor to the...
9.
Hovhannisyan Y, Melikyan G, Mougenot N, Gao-Li J, Friguet B, Paulin D, et al.
PLoS One
. 2019 Apr;
14(4):e0215821.
PMID: 31013315
Previous studies have shown that proteasome inhibition can have beneficial effects in dystrophic mouse models. In this study, we have investigated the effects of a new selective proteasome inhibitor, CLi,...
10.
Trellu S, Courties A, Jaisson S, Gorisse L, Gillery P, Kerdine-Romer S, et al.
Arthritis Res Ther
. 2019 Jan;
21(1):18.
PMID: 30635030
Background: Accumulation of advanced glycation end-products (AGEs) is involved in age-related osteoarthritis (OA). Glyoxalase (Glo)-1 is the main enzyme involved in the removal of AGE precursors, especially carboxymethyl-lysine (CML). We...