Bernardo Moreno-Lopez
Overview
Explore the profile of Bernardo Moreno-Lopez including associated specialties, affiliations and a list of published articles.
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27
Citations
499
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Recent Articles
11.
Montero F, Sunico C, Liu B, Paton J, Kasparov S, Moreno-Lopez B
J Physiol
. 2010 Jul;
588(Pt 18):3425-43.
PMID: 20660560
Dysregulation of protein expression, function and/or aggregation is a hallmark of a number of neuropathological conditions. Among them, upregulation and/or de novo expression of the neuronal isoform of nitric oxide...
12.
Sunico C, Dominguez G, Garcia-Verdugo J, Osta R, Montero F, Moreno-Lopez B
Brain Pathol
. 2010 Jul;
21(1):1-15.
PMID: 20653686
Excitotoxicity is a widely studied mechanism underlying motoneuron degeneration in amyotrophic lateral sclerosis (ALS). Synaptic alterations that produce an imbalance in the ratio of inhibitory/excitatory synapses are expected to promote...
13.
Cantero J, Moreno-Lopez B, Portillo F, Rubio A, Hita-Yanez E, Avila J
Hippocampus
. 2010 Jul;
21(8):827-34.
PMID: 20607795
Tau is a neuronal microtubule-associated protein implicated in microtubules stabilization, axonal establishment and elongation during neuronal morphogenesis. Because of its elevated expression in neocortical regions and hippocampus, tau might play...
14.
Cantero J, Hita-Yanez E, Moreno-Lopez B, Portillo F, Rubio A, Avila J
J Alzheimers Dis
. 2010 Jun;
21(2):411-21.
PMID: 20555133
Evidence has shown that the lack of tau produces subtle changes in neuronal structure and modest impairment in complex behaviors, suggesting compensatory mechanisms carried out by other neuronal microtubule-associated proteins....
15.
Moreno-Lopez B
J Neurosci Res
. 2010 Feb;
88(9):1846-57.
PMID: 20143424
Physical injury to a nerve is the most frequent cause of acquired peripheral neuropathy, which is responsible for loss of motor, sensory and/or autonomic functions. Injured axons in the peripheral...
16.
Sunico C, Moreno-Lopez B
Neurosci Lett
. 2010 Jan;
471(2):119-24.
PMID: 20093168
The loss of intimate contact with axons triggers Schwann cells (SCs) to switch from a myelin-producing phenotype to a dedifferentiated, proliferating non-myelin-forming state after nerve injury. SC dedifferentiation is required...
17.
Sunico C, Gonzalez-Forero D, Dominguez G, Garcia-Verdugo J, Moreno-Lopez B
J Neurosci
. 2010 Jan;
30(3):973-84.
PMID: 20089906
The molecular signaling that underpins synapse loss in neuropathological conditions remains unknown. Concomitant upregulation of the neuronal nitric oxide (NO) synthase (nNOS) in neurodegenerative processes places NO at the center...
18.
Montero F, Portillo F, Gonzalez-Forero D, Moreno-Lopez B
Eur J Neurosci
. 2008 Jul;
28(1):107-16.
PMID: 18616563
Motoneurons integrate interneuronal activity into commands for skeletal muscle contraction and relaxation to perform motor actions. Hypoglossal motoneurons (HMNs) are involved in essential motor functions such as breathing, mastication, swallowing...
19.
Romero-Grimaldi C, Moreno-Lopez B, Estrada C
J Comp Neurol
. 2007 Nov;
506(2):339-46.
PMID: 18022945
Nitric oxide (NO) synthase (NOS) is developmentally regulated in the embryonic brain, where NO participates in cell proliferation, survival, and differentiation. In adults, NO inhibits neurogenesis under physiological conditions. This...
20.
Gonzalez-Forero D, Portillo F, Gomez L, Montero F, Kasparov S, Moreno-Lopez B
J Neurosci
. 2007 Jun;
27(23):6302-12.
PMID: 17554004
Glutamate-induced excitotoxicity, the most common pathological mechanism leading to neuronal death, may occur even with normal levels of glutamate if it coincides with a persistent enhancement of neuronal excitability. Neurons...