Be Wieringa
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Explore the profile of Be Wieringa including associated specialties, affiliations and a list of published articles.
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76
Citations
2333
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Recent Articles
11.
Xicoy H, Wieringa B, Martens G
Mol Neurodegener
. 2017 Jan;
12(1):10.
PMID: 28118852
Parkinson's disease (PD) is a devastating and highly prevalent neurodegenerative disease for which only symptomatic treatment is available. In order to develop a truly effective disease-modifying therapy, improvement of our...
12.
Gudde A, van Heeringen S, de Oude A, van Kessel I, Estabrook J, Wang E, et al.
RNA Biol
. 2017 Jan;
14(10):1374-1388.
PMID: 28102759
The unstable (CTG·CAG)n trinucleotide repeat in the myotonic dystrophy type 1 (DM1) locus is bidirectionally transcribed from genes with terminal overlap. By transcription in the sense direction, the DMPK gene...
13.
Gudde A, Gonzalez-Barriga A, van den Broek W, Wieringa B, Wansink D
Hum Mol Genet
. 2016 Feb;
25(8):1648-62.
PMID: 26908607
Muscular manifestation of myotonic dystrophy type 1 (DM1), a common inheritable degenerative multisystem disorder, is mainly caused by expression of RNA from a (CTG·CAG)n-expanded DM1 locus. Here, we report on...
14.
Gonzalez-Barriga A, Kranzen J, Croes H, Bijl S, van den Broek W, van Kessel I, et al.
PLoS One
. 2015 Mar;
10(3):e0121556.
PMID: 25799359
Myotonic Dystrophy type 1 (DM1) is a multisystemic disease caused by toxic RNA from a DMPK gene carrying an expanded (CTG•CAG)n repeat. Promising strategies for treatment of DM1 patients are...
15.
Venter G, Polling S, Pluk H, Venselaar H, Wijers M, Willemse M, et al.
Eur J Cell Biol
. 2014 Dec;
94(2):114-27.
PMID: 25538032
Subcellular partitioning of creatine kinase contributes to the formation of patterns in intracellular ATP distribution and the fuelling of cellular processes with a high and sudden energy demand. We have...
16.
Venter G, Oerlemans F, Willemse M, Wijers M, Fransen J, Wieringa B
PLoS One
. 2014 May;
9(5):e97378.
PMID: 24824795
Functional morphodynamic behavior of differentiated macrophages is strongly controlled by actin cytoskeleton rearrangements, a process in which also metabolic cofactors ATP and NAD(H) (i.e. NAD+ and NADH) and NADP(H) (i.e....
17.
Venter G, Oerlemans F, Wijers M, Willemse M, Fransen J, Wieringa B
PLoS One
. 2014 May;
9(5):e96786.
PMID: 24796786
Macrophages constantly undergo morphological changes when quiescently surveying the tissue milieu for signs of microbial infection or damage, or after activation when they are phagocytosing cellular debris or foreign material....
18.
Gonzalez-Barriga A, Mulders S, van de Giessen J, Hooijer J, Bijl S, van Kessel I, et al.
Mol Ther Nucleic Acids
. 2013 Mar;
2:e81.
PMID: 23511335
Myotonic dystrophy type 1 (DM1) is caused by DM protein kinase (DMPK) transcripts containing an expanded (CUG)n repeat. Antisense oligonucleotide (AON)-mediated suppression of these mutant RNAs is considered a promising...
19.
van Horssen R, Willemse M, Haeger A, Attanasio F, Guneri T, Schwab A, et al.
Cell Mol Life Sci
. 2013 Jan;
70(12):2175-90.
PMID: 23307072
Oncogenic transformation involves reprogramming of cell metabolism, whereby steady-state levels of intracellular NAD(+) and NADH can undergo dramatic changes while ATP concentration is generally well maintained. Altered expression of nicotinamide...
20.
van Horssen R, Buccione R, Willemse M, Cingir S, Wieringa B, Attanasio F
Eur J Cell Biol
. 2013 Jan;
92(3):113-21.
PMID: 23306026
Transformed cancer cells have an altered metabolism, characterized by a shift towards aerobic glycolysis, referred to as 'the Warburg phenotype'. A change in flux through mitochondrial OXPHOS and cytosolic pathways...