Andrew J Whatmore
Overview
Explore the profile of Andrew J Whatmore including associated specialties, affiliations and a list of published articles.
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10
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69
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Recent Articles
1.
Westwood M, Tajbakhsh S, Siddals K, Whatmore A, Clayton P
Pediatr Res
. 2011 Mar;
70(1):25-30.
PMID: 21430600
Girls with Turner syndrome (TS) are treated with supraphysiological doses of growth hormone (GH) to improve final height; however in some girls, the growth response can be poor. This may...
2.
Westwood M, Maqsood A, Solomon M, Whatmore A, Davis J, Baxter R, et al.
Am J Physiol Endocrinol Metab
. 2009 Oct;
298(3):E467-76.
PMID: 19861588
Normal childhood growth is determined by ultradian and infradian variations in GH secretion, yet GH treatment of children with short stature is restricted to daily fixed doses. We have used...
3.
Whatmore A, Patel L, Clayton P
Clin Endocrinol (Oxf)
. 2008 Nov;
70(3):429-34.
PMID: 19018778
Response to GH treatment is variable and dependent on diagnosis and dose. We used a pharmacogenomic approach to assess whether this variability is reflected in patterns of GH-induced gene expression...
4.
Maqsood A, Trueman J, Whatmore A, Westwood M, Price D, Hall C, et al.
Horm Res
. 2007 Mar;
68(5):225-30.
PMID: 17389812
Background/aims: Leptin is necessary for normal human pubertal development but its exact role in the period leading up to the onset of puberty has not been defined. This study has...
5.
Patel L, Cavazzoni E, Whatmore A, Carney S, Wales J, Clayton P, et al.
Pediatr Res
. 2007 Jan;
61(1):99-104.
PMID: 17211149
We determined the contributions of IGF-I, IGFBP-3 and leptin to growth in extremely premature infants over the first two years. Weight (Wt), crown-to heel length (CHL), plasma IGF-I, IGFBP-3 and...
6.
Banerjee I, Trueman J, Hall C, Price D, Patel L, Whatmore A, et al.
Eur J Endocrinol
. 2006 Jun;
155(1):121-6.
PMID: 16793957
Objectives: Constitutional delay of growth and puberty (CDGP) is a variant of normal pubertal timing and progress, often with dominant inheritance. It is likely that one or more genes will...
7.
Alba M, Hall C, Whatmore A, Clayton P, Price D, Salvatori R
Clin Endocrinol (Oxf)
. 2004 Mar;
60(4):470-5.
PMID: 15049962
Objective: Mutations in the GHRH receptor (GHRHR) gene (GHRHR) cause autosomal recessive isolated GH deficiency (IGHD), and are usually associated with anterior pituitary hypoplasia (APH) (defined as pituitary height more...
8.
Carakushansky M, Whatmore A, Clayton P, Shalet S, Gleeson H, Price D, et al.
Eur J Endocrinol
. 2003 Jan;
148(1):25-30.
PMID: 12534354
Objective: Mutations in the GH-releasing hormone (GHRH) receptor (GHRHR) gene (GHRHR) cause autosomal recessive familial isolated GH deficiency (IGHD). We searched for GHRHR mutations in two siblings with IGHD type...
9.
Zaman N, Hall C, Gill M, Jones J, Tillmann V, Westwood M, et al.
Clin Endocrinol (Oxf)
. 2003 Jan;
58(1):78-85.
PMID: 12519416
Objective: Leptin has been implicated in the interaction between nutrition, energy balance and sexual maturation in humans. A non-invasive method of measuring leptin would greatly facilitate longitudinal studies of changes...
10.
Silva C, Kloth M, Whatmore A, Freeth J, Anderson N, Laughlin K, et al.
Endocrinology
. 2002 Jun;
143(7):2610-7.
PMID: 12072393
We have investigated and compared GH and epidermal growth factor (EGF) signaling in primary human skin fibroblasts from normal subjects and subjects with GH-binding protein-positive Laron syndrome (LS). In normal...