Andrew D Wells
Overview
Explore the profile of Andrew D Wells including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
105
Citations
3860
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Trang K, Pahl M, Pippin J, Su C, Littleton S, Sharma P, et al.
Elife
. 2025 Jan;
13.
PMID: 39813287
The prevalence of childhood obesity is increasing worldwide, along with the associated common comorbidities of type 2 diabetes and cardiovascular disease in later life. Motivated by evidence for a strong...
2.
Thakur R, Xu M, Sowards H, Yon J, Jessop L, Myers T, et al.
medRxiv
. 2025 Jan;
PMID: 39802764
Genome-wide association studies (GWAS) of melanoma risk have identified 68 independent signals at 54 loci. For most loci, specific functional variants and their respective target genes remain to be established....
3.
Hart A, Kotzin J, Schulz S, Dunham J, Keenan A, Baker J, et al.
JCI Insight
. 2024 Dec;
10(3).
PMID: 39688922
In systemic lupus erythematosus (lupus), environmental effects acting within a permissive genetic background lead to autoimmune dysregulation. Dysfunction of CD4+ T cells contributes to pathology by providing help to autoreactive...
4.
Zhong J, OBrien A, Patel M, Eiser D, Mobaraki M, Collins I, et al.
medRxiv
. 2024 Oct;
PMID: 39371173
Identification of somatic driver mutations in the noncoding genome remains challenging. To comprehensively characterize noncoding driver mutations for pancreatic ductal adenocarcinoma (PDAC), we first created genome-scale maps of accessible chromatin...
5.
Pahl M, Sharma P, Thomas R, Thompson Z, Mount Z, Pippin J, et al.
Elife
. 2024 Sep;
13.
PMID: 39302339
Genome-wide association studies (GWAS) have identified hundreds of genetic signals associated with autoimmune disease. The majority of these signals are located in non-coding regions and likely impact -regulatory elements (cRE)....
6.
Wachowski N, Pippin J, Boehm K, Lu S, Leonard M, Manduchi E, et al.
Diabetologia
. 2024 Sep;
67(12):2740-2753.
PMID: 39240351
Aims/hypothesis: Genome-wide association studies (GWAS) have identified hundreds of type 2 diabetes loci, with the vast majority of signals located in non-coding regions; as a consequence, it remains largely unclear...
7.
Burton E, Argenziano M, Cook K, Ridler M, Lu S, Su C, et al.
bioRxiv
. 2024 Sep;
PMID: 39229212
Late-onset Alzheimer's disease (LOAD) research has principally focused on neurons over the years due to their known role in the production of amyloid beta plaques and neurofibrillary tangles. In contrast,...
8.
Trang K, Sharma P, Cook L, Mount Z, Thomas R, Kulkarni N, et al.
medRxiv
. 2024 Aug;
PMID: 39185517
A portion of the genetic basis for many common autoimmune disorders has been uncovered by genome-wide association studies (GWAS), but GWAS do not reveal causal variants, effector genes, or the...
9.
Trang K, Chesi A, Toikumo S, Pippin J, Pahl M, OBrien J, et al.
medRxiv
. 2024 Jul;
PMID: 39072016
Recent genome-wide association studies (GWAS) have revealed shared genetic components among alcohol, opioid, tobacco and cannabis use disorders. However, the extent of the underlying shared causal variants and effector genes,...
10.
Littleton S, Trang K, Volpe C, Cook K, DeBruyne N, Maguire J, et al.
Cell Genom
. 2024 May;
4(5):100556.
PMID: 38697123
The ch12q13 locus is among the most significant childhood obesity loci identified in genome-wide association studies. This locus resides in a non-coding region within FAIM2; thus, the underlying causal variant(s)...