Agnesska C Benjamin
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Explore the profile of Agnesska C Benjamin including associated specialties, affiliations and a list of published articles.
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11
Citations
468
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Recent Articles
1.
Horsnell H, Tetley R, De Belly H, Makris S, Millward L, Benjamin A, et al.
Nat Immunol
. 2022 Jul;
23(8):1169-1182.
PMID: 35882934
Emergent physical properties of tissues are not readily understood by reductionist studies of their constituent cells. Here, we show molecular signals controlling cellular, physical, and structural properties and collectively determine...
2.
de Winde C, George S, Crosas-Molist E, Hari-Gupta Y, Arp A, Benjamin A, et al.
iScience
. 2021 Sep;
24(9):102976.
PMID: 34485858
Melanoma is an aggressive skin cancer developing from melanocytes, frequently resulting in metastatic disease. Melanoma cells utilize amoeboid migration as mode of local invasion. Amoeboid invasion is characterized by rounded...
3.
de Winde C, Makris S, Millward L, Cantoral-Rebordinos J, Benjamin A, Martinez V, et al.
J Cell Sci
. 2021 Jun;
134(14).
PMID: 34184727
In adaptive immunity, CLEC-2+ dendritic cells (DCs) contact fibroblastic reticular cells (FRCs) inhibiting podoplanin-dependent actomyosin contractility, permitting FRC spreading and lymph node expansion. The molecular mechanisms controlling lymph node remodelling...
4.
Martinez V, Pankova V, Krasny L, Singh T, Makris S, White I, et al.
Cell Rep
. 2019 Nov;
29(9):2810-2822.e5.
PMID: 31775047
Lymph nodes (LNs) act as filters, constantly sampling peripheral cues. This is facilitated by the conduit network, a tubular structure of aligned extracellular matrix (ECM) fibrils ensheathed by fibroblastic reticular...
5.
Neueder A, Dumas A, Benjamin A, Bates G
Nat Commun
. 2018 Sep;
9(1):3955.
PMID: 30262848
Huntington's disease is caused by a CAG repeat expansion in exon 1 of the HTT gene. We have previously shown that exon 1 HTT does not always splice to exon...
6.
Pido-Lopez J, Andre R, Benjamin A, Ali N, Farag S, Tabrizi S, et al.
Sci Rep
. 2018 Aug;
8(1):11447.
PMID: 30061661
Neurodegenerative diseases, characterised by the progressive and selective neuronal death in the central nervous system, are frequently accompanied by an activated immune system. In Huntington's disease (HD), clinical and animal...
7.
Bondulich M, Jolinon N, Osborne G, Smith E, Rattray I, Neueder A, et al.
Sci Rep
. 2017 Oct;
7(1):14275.
PMID: 29079832
Huntington's disease (HD) is an inherited neurodegenerative disorder of which skeletal muscle atrophy is a common feature, and multiple lines of evidence support a muscle-based pathophysiology in HD mouse models....
8.
Bryant D, Landles C, Papadopoulou A, Benjamin A, Duckworth J, Rosahl T, et al.
Sci Rep
. 2017 Sep;
7(1):11900.
PMID: 28928414
Histone Deacetylase 11 (HDAC11) is highly expressed in the central nervous system where it has been reported to have roles in neural differentiation. In contrast with previous studies showing nuclear...
9.
Tulino R, Benjamin A, Jolinon N, Smith D, Chini E, Carnemolla A, et al.
PLoS One
. 2016 Feb;
11(2):e0150682.
PMID: 26919088
[This corrects the article DOI: 10.1371/journal.pone.0145425.].
10.
Tulino R, Benjamin A, Jolinon N, Smith D, Chini E, Carnemolla A, et al.
PLoS One
. 2016 Jan;
11(1):e0145425.
PMID: 26815359
Huntington's disease (HD) is a neurodegenerative disorder for which there are no disease-modifying treatments. SIRT1 is a NAD+-dependent protein deacetylase that is implicated in maintaining neuronal health during development, differentiation...