Andreas Neueder
Overview
Explore the profile of Andreas Neueder including associated specialties, affiliations and a list of published articles.
Author names and details appear as published. Due to indexing inconsistencies, multiple individuals may share a name, and a single author may have variations. MedLuna displays this data as publicly available, without modification or verification
Snapshot
Snapshot
Articles
31
Citations
1024
Followers
0
Related Specialties
Related Specialties
Top 10 Co-Authors
Top 10 Co-Authors
Published In
Published In
Affiliations
Affiliations
Soon will be listed here.
Recent Articles
1.
Heiden R, Hannig L, Bernhard J, Vallon M, Schlecht A, Hofmann N, et al.
iScience
. 2025 Feb;
28(2):111740.
PMID: 39925414
Tight junctions of vascular endothelial cells in the central nervous system form the blood-brain and inner blood-retinal barriers, the integrity of which are further influenced by neighboring cells such as...
2.
Hoschek F, Natan J, Wagner M, Sathasivam K, Abdelmoez A, Einem B, et al.
Mol Med
. 2024 Apr;
30(1):49.
PMID: 38600455
No abstract available.
3.
Hoschek F, Natan J, Wagner M, Sathasivam K, Abdelmoez A, Einem B, et al.
Mol Med
. 2024 Mar;
30(1):36.
PMID: 38459427
Background: The disease-causing mutation in Huntington disease (HD) is a CAG trinucleotide expansion in the huntingtin (HTT) gene. The mutated CAG tract results in the production of a small RNA,...
4.
Neueder A, Kojer K, Gu Z, Wang Y, Hering T, Tabrizi S, et al.
Brain
. 2024 Jan;
147(6):2009-2022.
PMID: 38195181
Huntington's disease (HD) predominantly affects the brain, causing a mixed movement disorder, cognitive decline and behavioural abnormalities. It also causes a peripheral phenotype involving skeletal muscle. Mitochondrial dysfunction has been...
5.
Neueder A, Nitzschner P, Wagner R, Hummel J, Hoschek F, Wagner M, et al.
Clin Transl Med
. 2024 Jan;
14(1):e1525.
PMID: 38193625
No abstract available.
6.
Lange J, Gillham O, Flower M, Ging H, Eaton S, Kapadia S, et al.
Prog Neurobiol
. 2023 Apr;
225:102448.
PMID: 37023937
Huntington's Disease (HD) is a neurodegenerative disease caused by a polyglutamine (polyQ) expansion in the Huntingtin gene. Astrocyte dysfunction is known to contribute to HD pathology, however our understanding of...
7.
Neueder A, Kojer K, Hering T, Lavery D, Chen J, Birth N, et al.
Genome Biol
. 2022 Sep;
23(1):189.
PMID: 36071529
Background: A major challenge in neurodegenerative diseases concerns identifying biological disease signatures that track with disease progression or respond to an intervention. Several clinical trials in Huntington disease (HD), an...
8.
Bielmeier C, Schmitt S, Kleefeldt N, Boneva S, Schlecht A, Vallon M, et al.
Int J Mol Sci
. 2022 Mar;
23(5).
PMID: 35269767
Transforming growth factor β (TGFβ) signaling has manifold functions such as regulation of cell growth, differentiation, migration, and apoptosis. Moreover, there is increasing evidence that it also acts in a...
9.
Sonmez A, Mustafa R, Ryll S, Tuorto F, Wacheul L, Ponti D, et al.
Cell Death Dis
. 2021 Dec;
12(12):1139.
PMID: 34880223
Transcriptional and cellular-stress surveillance deficits are hallmarks of Huntington's disease (HD), a fatal autosomal-dominant neurodegenerative disorder caused by a pathological expansion of CAG repeats in the Huntingtin (HTT) gene. The...
10.
Petrozziello T, Bordt E, Mills A, Kim S, Sapp E, Devlin B, et al.
Mol Neurobiol
. 2021 Nov;
59(1):683-702.
PMID: 34757590
Understanding the mechanisms underlying amyotrophic lateral sclerosis (ALS) is crucial for the development of new therapies. Previous studies have demonstrated that mitochondrial dysfunction is a key pathogenetic event in ALS....