Aaron B Bradford
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Explore the profile of Aaron B Bradford including associated specialties, affiliations and a list of published articles.
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7
Citations
52
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Recent Articles
1.
Machamer J, Vazquez-Cintron E, Stenslik M, Pagarigan K, Bradford A, Ondeck C, et al.
Front Cell Neurosci
. 2023 Aug;
17:1226194.
PMID: 37650071
Introduction: Botulinum neurotoxin (BoNT) causes neuroparalytic disease and death by blocking neuromuscular transmission. There are no specific therapies for clinical botulism and the only treatment option is supportive care until...
2.
Bradford A, Machamer J, Russo T, McNutt P
Toxicol Appl Pharmacol
. 2018 Jan;
341:77-86.
PMID: 29366638
Botulinum neurotoxins (BoNTs) are exceedingly potent neurological poisons that prevent neurotransmitter release from peripheral nerve terminals by cleaving presynaptic proteins required for synaptic vesicle fusion. The ensuing neuromuscular paralysis causes...
3.
Beske P, Bradford A, Hoffman K, Mason S, McNutt P
Toxicon
. 2017 Oct;
147:47-53.
PMID: 29054436
Botulinum neurotoxins (BoNTs) are exceedingly potent neurological poisons that block cholinergic release in the peripheral nervous system and cause death by asphyxiation. While post-exposure prophylaxis can effectively eliminate toxin in...
4.
Beske P, Bradford A, Grynovicki J, Glotfelty E, Hoffman K, Hubbard K, et al.
Toxicol Sci
. 2015 Nov;
149(2):503-15.
PMID: 26615023
Clinical manifestations of tetanus and botulism result from an intricate series of interactions between clostridial neurotoxins (CNTs) and nerve terminal proteins that ultimately cause proteolytic cleavage of SNARE (soluble N-ethylmaleimide-sensitive...
5.
Bradford A, Mancini J, Atchison W
J Pharmacol Exp Ther
. 2015 Oct;
356(1):2-12.
PMID: 26514794
Methylmercury (MeHg) disrupts cerebellar function, especially during development. Cerebellar granule cells (CGC), which are particularly susceptible to MeHg by unknown mechanisms, migrate during this process. Transient changes in intracellular Ca(2+)...
6.
Bradford A, McNutt P
World J Stem Cells
. 2015 Aug;
7(6):899-921.
PMID: 26240679
Functional synaptogenesis and network emergence are signature endpoints of neurogenesis. These behaviors provide higher-order confirmation that biochemical and cellular processes necessary for neurotransmitter release, post-synaptic detection and network propagation of...
7.
Neal A, Molina-Campos E, Marrero-Rosado B, Bradford A, Fox S, Kovalova N, et al.
J Neurosci
. 2010 Feb;
30(8):2807-9.
PMID: 20181577
No abstract available.