Askari A
Pharmacol Res Perspect. 2019; 7(4):e00505.
PMID: 31360524
PMC: 6639696.
DOI: 10.1002/prp2.505.
Rao D, Liu H, Ambudkar S, Mayer M
J Biol Chem. 2014; 289(45):31397-410.
PMID: 25253691
PMC: 4223339.
DOI: 10.1074/jbc.M114.576819.
Mirams G, Davies M, Cui Y, Kohl P, Noble D
Br J Pharmacol. 2012; 167(5):932-45.
PMID: 22568589
PMC: 3492977.
DOI: 10.1111/j.1476-5381.2012.02020.x.
Calabrese E
Br J Clin Pharmacol. 2008; 66(5):594-617.
PMID: 18662293
PMC: 2661975.
DOI: 10.1111/j.1365-2125.2008.03243.x.
ORourke B, Maack C
Drug Discov Today Dis Models. 2008; 4(4):207-217.
PMID: 18650959
PMC: 2440714.
DOI: 10.1016/j.ddmod.2007.11.003.
Isoform-specific stimulation of cardiac Na/K pumps by nanomolar concentrations of glycosides.
Gao J, Wymore R, Wang Y, Gaudette G, Krukenkamp I, Cohen I
J Gen Physiol. 2002; 119(4):297-312.
PMID: 11929882
PMC: 2238186.
DOI: 10.1085/jgp.20028501.
Low concentrations of ouabain stimulate Na/Ca exchange in neurons.
Saghian A, Ayrapetyan S, Carpenter D
Cell Mol Neurobiol. 1996; 16(4):489-98.
PMID: 8879751
PMC: 11563144.
DOI: 10.1007/BF02150229.
In vivo assessment of the inotropic and toxic effects of oxidized ouabain.
Arad M, Shotan A, Heller M, Rabinowitz B, Uretzki G
Basic Res Cardiol. 1993; 88(1):42-51.
PMID: 8471002
DOI: 10.1007/BF00788529.
Digoxin activates sarcoplasmic reticulum Ca(2+)-release channels: a possible role in cardiac inotropy.
McGarry S, Williams A
Br J Pharmacol. 1993; 108(4):1043-50.
PMID: 8387382
PMC: 1908139.
DOI: 10.1111/j.1476-5381.1993.tb13503.x.
Characterization of the inotropic and arrhythmogenic action of the sodium channel activator BDF 9148: a comparison to its S-enantiomer BDF 9196, to its congener DPI 201-106, to norepinephrine, and to ouabain.
Baumgart D, Ehring T, Krajcar M, Skyschally A, Heusch G
Basic Res Cardiol. 1994; 89(1):61-79.
PMID: 8010937
DOI: 10.1007/BF00788678.
Stimulation of monovalent cation active transport by low concentrations of cardiac glycosides. Role of catecholamines.
Hougen T, Spicer N, Smith T
J Clin Invest. 1981; 68(5):1207-14.
PMID: 7298847
PMC: 370915.
DOI: 10.1172/jci110366.
Effect of strophanthidin on intracellular Na ion activity and twitch tension of constantly driven canine cardiac Purkinje fibers.
Lee C, DAgostino M
Biophys J. 1982; 40(3):185-98.
PMID: 7183333
PMC: 1328995.
DOI: 10.1016/S0006-3495(82)84474-3.
The effects of low concentrations of cardiotonic steroids on membrane currents and tension in sheep Purkinje fibres.
Hart G, Noble D, Shimoni Y
J Physiol. 1983; 334:103-31.
PMID: 6864554
PMC: 1197303.
DOI: 10.1113/jphysiol.1983.sp014483.
Electrophysiological studies of some semisynthetic cardiac glycoside derivatives in isolated papillary muscle of the guinea-pig.
LULLMANN H, Niehus U, Pulss W, Ravens U
Br J Pharmacol. 1983; 79(3):755-64.
PMID: 6652355
PMC: 2044912.
DOI: 10.1111/j.1476-5381.1983.tb10014.x.
Inhibition of Na+/Ca2+ exchange in membrane vesicle and papillary muscle preparations from guinea pig heart by analogs of amiloride.
Siegl P, Cragoe Jr E, Trumble M, Kaczorowski G
Proc Natl Acad Sci U S A. 1984; 81(10):3238-42.
PMID: 6587348
PMC: 345257.
DOI: 10.1073/pnas.81.10.3238.
The inotropic effect of ouabain and its antagonism by dihydroouabain in rat isolated atria and ventricles in relation to specific binding sites.
Finet M, GODFRAIND T, Noel F
Br J Pharmacol. 1983; 80(4):751-9.
PMID: 6571229
PMC: 2045062.
DOI: 10.1111/j.1476-5381.1983.tb10067.x.
Digitalis intoxication: a new approach to an old problem.
George C
Br Med J (Clin Res Ed). 1983; 286(6377):1533-4.
PMID: 6405875
PMC: 1547999.
DOI: 10.1136/bmj.286.6377.1533.
Excitation-contraction coupling in cardiac Purkinje fibers. Effects of cardiotonic steroids on the intracellular [Ca2+] transient, membrane potential, and contraction.
Wier W, Hess P
J Gen Physiol. 1984; 83(3):395-415.
PMID: 6325588
PMC: 2215639.
DOI: 10.1085/jgp.83.3.395.
The relationship among intracellular sodium activity, calcium, and strophanthidin inotropy in canine cardiac Purkinje fibers.
Vassalle M, Lee C
J Gen Physiol. 1984; 83(2):287-307.
PMID: 6325584
PMC: 2215632.
DOI: 10.1085/jgp.83.2.287.
Can blocking the Na/K exchange pump lead to a reduction in intracellular sodium?.
Cohen I
Experientia. 1983; 39(11):1280-2.
PMID: 6315475
DOI: 10.1007/BF01990370.