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A Bcl-xL Timer Sets Platelet Life Span

Overview
Journal Cell
Publisher Cell Press
Specialty Cell Biology
Date 2007 Mar 27
PMID 17382877
Citations 11
Authors
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Abstract

Platelets are cell fragments lacking nuclei that play a key role in blood clotting. Using an impressive genetic screen involving ENU-mutagenesis of whole mice, Mason et al. (2007) report in this issue their identification of mutations in the antiapoptotic protein Bcl-x(L) that cause accelerated death of platelets leading to platelet deficiency.

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