Yoshiyuki Seki
Overview
Explore the profile of Yoshiyuki Seki including associated specialties, affiliations and a list of published articles.
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Articles
22
Citations
1044
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Recent Articles
1.
Yamamoto M, Suwa Y, Sugiyama K, Okashita N, Kawaguchi M, Tani N, et al.
J Cell Sci
. 2020 Jul;
133(15).
PMID: 32661086
The pluripotency-associated transcriptional network is regulated by a core circuitry of transcription factors. The PR domain-containing protein PRDM14 maintains pluripotency by activating and repressing transcription in a target gene-dependent manner....
2.
Shibata Y, Seki Y, Nishiwaki K
Biol Open
. 2019 Jan;
8(1).
PMID: 30635266
Cell-fate maintenance is important to preserve the variety of cell types that are essential for the formation and function of tissues. We previously showed that the acetylated histone-binding protein BET-1...
3.
Kawaguchi M, Sugiyama K, Matsubara K, Lin C, Kuraku S, Hashimoto S, et al.
Development
. 2019 Jan;
146(2).
PMID: 30630825
Gene regulatory networks underlying cellular pluripotency are controlled by a core circuitry of transcription factors in mammals, including POU5F1. However, the evolutionary origin and transformation of pluripotency-related transcriptional networks have...
4.
Seki Y
Front Cell Dev Biol
. 2018 Mar;
6:12.
PMID: 29487849
PR-domain containing protein 14 (PRDM14) is a site-specific DNA-binding protein and is required for establishment of pluripotency in embryonic stem cells (ESCs) and primordial germ cells (PGCs) in mice. DNA...
5.
Okashita N, Suwa Y, Nishimura O, Sakashita N, Kadota M, Nagamatsu G, et al.
Stem Cell Reports
. 2016 Nov;
7(6):1072-1086.
PMID: 27866876
Primordial germ cells (PGCs) are specified from epiblast cells in mice. Genes associated with naive pluripotency are repressed in the transition from inner cell mass to epiblast cells, followed by...
6.
Okashita N, Sakashita N, Ito K, Mitsuya A, Suwa Y, Seki Y
Biochem Biophys Res Commun
. 2015 Sep;
466(1):138-45.
PMID: 26325469
Pluripotency and self-renewal of mouse embryonic stem cells (ESCs) depend on a network of transcription factors maintained by exogenous leukaemia inhibitory factor (LIF). PR-domain containing transcriptional regulator 14 (PRDM14), is...
7.
Okashita N, Kumaki Y, Ebi K, Nishi M, Okamoto Y, Nakayama M, et al.
Development
. 2013 Dec;
141(2):269-80.
PMID: 24335252
Ten-eleven translocation (TET) proteins oxidize 5-methylcytosine (5mC) to 5-hydroxymethylcytosine (5hmC), 5-formylcytosine (5fC) and 5-carboxylcytosine (5caC). 5fC and 5caC can be excised and repaired by the base excision repair (BER) pathway,...
8.
A replication-dependent passive mechanism modulates DNA demethylation in mouse primordial germ cells
Ohno R, Nakayama M, Naruse C, Okashita N, Takano O, Tachibana M, et al.
Development
. 2013 Jun;
140(14):2892-903.
PMID: 23760957
Germline cells reprogramme extensive epigenetic modifications to ensure the cellular totipotency of subsequent generations and to prevent the accumulation of epimutations. Notably, primordial germ cells (PGCs) erase genome-wide DNA methylation...
9.
Nakashima H, Kimura T, Kaga Y, Nakatani T, Seki Y, Nakamura T, et al.
Biol Reprod
. 2013 Apr;
88(5):125.
PMID: 23595900
DNA methylation is a central epigenetic event that regulates cellular differentiation, reprogramming, and pathogenesis. Genomewide DNA demethylation occurs in preimplantation embryos and in embryonic germ cell precursors called primordial germ...
10.