Walter Giaretti
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Explore the profile of Walter Giaretti including associated specialties, affiliations and a list of published articles.
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Articles
27
Citations
395
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Recent Articles
1.
Castagnola P, Gandolfo S, Malacarne D, Aiello C, Marino R, Zoppoli G, et al.
PLoS One
. 2017 Sep;
12(9):e0184425.
PMID: 28877236
The aim of this study was to investigate the relationship between tobacco smoke habit, patient age, DNA aneuploidy and genomic DNA copy number aberrations (CNAs) in oral potentially malignant disorder...
2.
Amaro A, Angelini G, Mirisola V, Esposito A, Reverberi D, Matis S, et al.
Oncotarget
. 2016 Sep;
7(42):68803-68820.
PMID: 27626697
The acquisition of an invasive phenotype is a prerequisite for metastasization, yet it is not clear whether or to which extent the invasive phenotype is linked to other features characteristic...
3.
Castagnola P, Zoppoli G, Gandolfo S, Monticone M, Malacarne D, Cirmena G, et al.
PLoS One
. 2015 Nov;
10(11):e0142294.
PMID: 26540282
Oral potentially malignant disorders (OPMDs) characterized by the presence of dysplasia and DNA copy number aberrations (CNAs), may reflect chromosomal instability (CIN) and predispose to oral squamous cell carcinoma (OSCC)....
4.
Maffei M, Mongera S, Terpstra L, Donadini A, Voorham Q, Meijer G, et al.
Cytogenet Genome Res
. 2014 Oct;
144(1):9-14.
PMID: 25323042
DNA aneuploid sublines in sporadic colorectal cancers (CRCs) are quite frequent (about 85%) and likely the consequence of chromosomal instability and DNA copy number aberrations (CNAs). In order to gain...
5.
Monticone M, Taherian R, Stigliani S, Carra E, Monteghirfo S, Longo L, et al.
PLoS One
. 2014 Mar;
9(2):e90085.
PMID: 24587218
Reactive oxygen species (ROS) are metabolism by-products that may act as signaling molecules to sustain tumor growth. Antioxidants have been used to impair cancer cell survival. Our goal was to...
6.
Giaretti W, Monteghirfo S, Pentenero M, Gandolfo S, Malacarne D, Castagnola P
Cancer Epidemiol Biomarkers Prev
. 2013 May;
22(6):1133-41.
PMID: 23629518
Background: Chromosomal instability and aneuploidy may represent biomarkers of oral exposure to damaging agents and early signs of clinical disease according to the theory of "oral field cancerization." Methods: The...
7.
Giaretti W, Pentenero M, Gandolfo S, Castagnola P
Future Oncol
. 2012 Nov;
8(10):1257-71.
PMID: 23130927
Carcinogen exposure of the oral cavity is thought to create an extensive 'field cancerization'. According to this model, a very early precursor of oral cancer is a patch of normal-appearing...
8.
Monticone M, Daga A, Candiani S, Romeo F, Mirisola V, Viaggi S, et al.
BMC Cancer
. 2012 Aug;
12:358.
PMID: 22901239
Background: Most patients affected by Glioblastoma multiforme (GBM, grade IV glioma) experience a recurrence of the disease because of the spreading of tumor cells beyond surgical boundaries. Unveiling mechanisms causing...
9.
Castagnola P, Malacarne D, Scaruffi P, Maffei M, Donadini A, Di Nallo E, et al.
BMC Cancer
. 2011 Oct;
11:445.
PMID: 21995418
Background: The mucosae of the oral cavity are different at the histological level but appear all equally exposed to common genotoxic agents. As a result of this exposure, changes in...
10.
Pentenero M, Donadini A, Di Nallo E, Maffei M, Marino R, Familiari U, et al.
J Oral Pathol Med
. 2011 Sep;
41(2):119-23.
PMID: 21950471
Objective: 'Field cancerization' is an accepted model for oral carcinogenesis. So far, genetically altered fields have been just reported in the presence of carcinomas. This study assessed the distant mirror...