W P BENNETT
Overview
Explore the profile of W P BENNETT including associated specialties, affiliations and a list of published articles.
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76
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2513
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Recent Articles
1.
Vahakangas K, BENNETT W, Castren K, Welsh J, Khan M, Blomeke B, et al.
Cancer Res
. 2001 Jun;
61(11):4350-6.
PMID: 11389059
Somatic p53 mutations are common in lung cancer. Active cigarette smoking is positively correlated with the total frequency of p53 mutations and G:C to T:A transversions on the nontranscribed (DNA...
2.
Feng J, Zheng J, Gelernter J, Kranzler H, Cook E, Goldman D, et al.
Mol Psychiatry
. 2001 Apr;
6(2):168-72.
PMID: 11317218
alpha(2) adrenergic receptors are activated by adrenaline and noradrenaline, and three subtypes (ie, A, B, C) have differential affinities for antagonists and medications. The alpha(2c) adrenergic receptor (ADRA2C), located on...
3.
Hussain S, Amstad P, Raja K, Ambs S, Nagashima M, BENNETT W, et al.
Cancer Res
. 2000 Jul;
60(13):3333-7.
PMID: 10910033
Ulcerative colitis (UC) is a chronic inflammatory disease that produces reactive oxygen and nitrogen species and increases the risk of colorectal cancer (CRC). The p53 tumor suppressor gene is frequently...
4.
Feng J, Zheng J, BENNETT W, Heston L, Jones I, Craddock N, et al.
Am J Med Genet
. 2000 Jul;
96(3):412-7.
PMID: 10898924
Steroid hormone administration causes behavior changes in many and psychosis in a few. The clinical features suggest that genetic variants of the glucocorticoid receptor or cofactors could produce susceptible subpopulations...
5.
Smith L, Denissenko M, BENNETT W, Li H, Amin S, Tang M, et al.
J Natl Cancer Inst
. 2000 May;
92(10):803-11.
PMID: 10814675
Background: Polycyclic aromatic hydrocarbons (PAHs) are ubiquitous in combustion products of organic matter, including cigarette smoke. Metabolically activated diol epoxides of these compounds, including benzo[a]pyrene diol epoxide (B[a]PDE), have been...
6.
Saito T, Krutovskikh V, Marion M, Ishak K, BENNETT W, Yamasaki H
Int J Cancer
. 2000 Mar;
86(1):67-70.
PMID: 10728596
Gap junctional intercellular communication is often impaired in cancers, and the genes which encode the connexin gap junction proteins are considered to be tumor-suppressor genes. In this study, we analyzed...
7.
BENNETT W, Alavanja M, Blomeke B, Vahakangas K, Castren K, Welsh J, et al.
J Natl Cancer Inst
. 1999 Dec;
91(23):2009-14.
PMID: 10580025
Background: Exposure to environmental tobacco smoke (ETS) is considered to be a major lung cancer risk factor for never smokers. We investigated the hypothesis that never-smoking women who are exposed...
8.
Rusin M, Butkiewicz D, Malusecka E, Zborek A, Harasim J, Czyzewski K, et al.
Br J Cancer
. 1999 Jul;
80(9):1445-52.
PMID: 10424749
The p53 mutation spectrum can generate hypotheses linking carcinogen exposure to human cancer. Although it is well-documented that tobacco smoking is a major cause of lung cancer, the contribution of...
9.
BENNETT W, Hussain S, Vahakangas K, Khan M, Shields P, Harris C
J Pathol
. 1999 May;
187(1):8-18.
PMID: 10341702
The p53 tumour suppressor gene is at the crossroads of a network of cellular pathways including cell cycle checkpoints, DNA repair, chromosomal segregation, and apoptosis. These pathways have evolved to...
10.
Przygodzki R, BENNETT W, Guinee Jr D, Khan M, FREEDMAN A, Shields P, et al.
Pharmacogenetics
. 1999 Jan;
8(6):503-11.
PMID: 9918134
p53 mutation status was analysed in relation to DNA polymorphisms of GSTM1, CYP1A1 and CYP2E1 from 105 surgically resected non-small cell lung cancer cases. Demographic factors, smoking, occupation, family history,...