Viswakalyan Kotapalli
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Explore the profile of Viswakalyan Kotapalli including associated specialties, affiliations and a list of published articles.
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10
Citations
79
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Recent Articles
1.
George S, Kotapalli V, Ramaswamy P, Kumar R, Gowrishankar S, Uppin S, et al.
J Cell Biochem
. 2024 Feb;
125(4):e30534.
PMID: 38358025
Missense mutations in the DNA binding domain of p53 are observed frequently in esophageal squamous cell carcinoma (ESCC). Recent studies have revealed the potentially oncogenic transcriptional networks regulated by mutant...
2.
Animireddy S, Kavadipula P, Kotapalli V, Gowrishankar S, Rao S, Bashyam M
J Cell Sci
. 2021 Jan;
134(4).
PMID: 33443092
The ARID1B (BAF250b) subunit of the human SWI/SNF chromatin remodeling complex is a canonical nuclear tumor suppressor. We employed prediction, intracellular fluorescence and cellular fractionation-based subcellular localization analyses to identify...
3.
Bala P, Singh A, Kavadipula P, Kotapalli V, Sabarinathan R, Bashyam M
Oncogene
. 2020 Dec;
40(4):863-874.
PMID: 33262464
Early-onset sporadic rectal cancer (EOSRC) is a unique and predominant colorectal cancer (CRC) subtype in India. In order to understand the tumorigenic process in EOSRC, we performed whole-exome sequencing of...
4.
Kumar R, Kotapalli V, Naz A, Gowrishankar S, Rao S, Pollack J, et al.
Genes Chromosomes Cancer
. 2018 Feb;
57(6):304-310.
PMID: 29383790
Canonical Wnt/β-catenin signaling plays important roles in embryonic development and adult tissue regeneration while aberrant Wnt activation is the major driver of sporadic colorectal cancer (CRC). Thus, it is important...
5.
Kumar R, Raman R, Kotapalli V, Gowrishankar S, Pyne S, Pollack J, et al.
J Mol Med (Berl)
. 2017 Nov;
96(2):135-146.
PMID: 29124284
Key Messages: Early-onset sporadic rectal cancer exhibits DNA gain and loss without Wnt activation. Ca/NFAT signaling appears to be activated in the absence of Wnt activation. An eight-gene genetic signature...
6.
Adduri R, Katamoni R, Pandilla R, Madana S, Paripati A, Kotapalli V, et al.
PLoS One
. 2014 Dec;
9(12):e114002.
PMID: 25436609
Purpose: The tumor suppressor p53 is known to be inactivated frequently in various cancers. In addition, germline polymorphisms in TP53 are known to affect protein function and influence risk of...
7.
Bashyam M, Kotapalli V, Raman R, Chaudhary A, Yadav B, Gowrishankar S, et al.
Mol Carcinog
. 2014 Nov;
54(12):1807-14.
PMID: 25420488
Lynch syndrome (LS), the most common form of familial CRC predisposition that causes tumor onset at a young age, is characterized by the presence of microsatellite instability (MSI) in tumors...
8.
Adduri Sr R, Kotapalli V, Gupta N, Gowrishankar S, Srinivasulu M, Ali M, et al.
BMC Clin Pathol
. 2014 Aug;
14:37.
PMID: 25152695
Background: Squamous cell carcinoma of tongue (SCCT) is expected to harbor unique clinico-pathological and molecular genetic features since a significant proportion of patients are young and exhibit no association with...
9.
Pandilla R, Kotapalli V, Gowrishankar S, Chigurupati M, Patnaik S, Uppin S, et al.
Eur J Clin Invest
. 2013 Oct;
43(12):1233-9.
PMID: 24102414
Background: The two main oesophageal cancer subtypes namely adenocarcinoma and squamous cell carcinoma exhibit interesting clinical, pathological and geographical variations with the former being more common in the West and...
10.
Raman R, Kotapalli V, Adduri R, Gowrishankar S, Bashyam L, Chaudhary A, et al.
Mol Carcinog
. 2012 Nov;
53 Suppl 1:E181-6.
PMID: 23168910
Two genetic instability pathways viz. chromosomal instability, driven primarily by APC mutation induced deregulated Wnt signaling, and microsatellite instability (MSI) caused by mismatch repair (MMR) inactivation, together account for >90%...