V Kaartinen
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Explore the profile of V Kaartinen including associated specialties, affiliations and a list of published articles.
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36
Citations
954
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Recent Articles
1.
Saroya G, Hu J, Hu M, Panaretos C, Mann J, Kim S, et al.
J Dent Res
. 2023 Feb;
102(4):459-466.
PMID: 36751050
Failure of palatogenesis results in cleft palate, one of the most common congenital disabilities in humans. During the final phases of palatogenesis, the protective function of the peridermal cell layer...
2.
Kaartinen V, Nagy A
Genesis
. 2001 Dec;
31(3):126-9.
PMID: 11747203
Conditional and tissue specific gene targeting using the Cre-loxP recombination system in combination with established ES cell techniques has become a standard for in vivo loss of function studies. In...
3.
Kaartinen V, Gonzalez-Gomez I, Voncken J, Haataja L, Faure E, Nagy A, et al.
Development
. 2001 Oct;
128(21):4217-27.
PMID: 11684658
Experiments in cultured cells have implicated the molecular switch Rac in a wide variety of cellular functions. Here we demonstrate that the simultaneous disruption of two negative regulators of Rac,...
4.
BLAVIER L, Lazaryev A, Groffen J, Heisterkamp N, DeClerck Y, Kaartinen V
Mol Biol Cell
. 2001 May;
12(5):1457-66.
PMID: 11359935
Cleft lip and palate syndromes are among the most common congenital malformations in humans. Mammalian palatogenesis is a complex process involving highly regulated interactions between epithelial and mesenchymal cells of...
5.
Hemmeryckx B, van Wijk A, Reichert A, Kaartinen V, de Jong R, Pattengale P, et al.
Cancer Res
. 2001 Mar;
61(4):1398-405.
PMID: 11245441
The adapter protein Crkl has been implicated in the abnormal signal transduction pathways activated by the Bcr/Abl oncoprotein, which causes Philadelphia-positive leukemias in humans. To investigate the role of Crkl...
6.
Faure E, Heisterkamp N, Groffen J, Kaartinen V
Cell Tissue Res
. 2000 May;
300(1):89-95.
PMID: 10805078
After cessation of lactation, the mammary gland undergoes involution, which is characterized by a massive epithelial cell death and proteolytic degradation of the extracellular matrix. Whereas the expression patterns and...
7.
Dunder U, Kaartinen V, Valtonen P, Vaananen E, Kosma V, Heisterkamp N, et al.
FASEB J
. 2000 Feb;
14(2):361-7.
PMID: 10657992
Aspartylglycosaminuria (AGU), the most common lysosomal disorder of glycoprotein degradation, is caused by deficient activity of glycosylasparaginase (AGA). AGA-deficient mice share most of the clinical, biochemical and histopathologic characteristics of...
8.
Shi W, Heisterkamp N, Groffen J, Zhao J, Warburton D, Kaartinen V
Am J Physiol
. 1999 Dec;
277(6):L1205-13.
PMID: 10600892
Newborn transforming growth factor (TGF)-beta3-null mutant mice exhibit defects of palatogenesis and pulmonary development. Glucocorticoids, which play a central role in fetal lung maturation, have been postulated to mediate their...
9.
Groffen J, de Jong R, Haataja L, Kaartinen V, Heisterkamp N
Leukemia
. 1999 May;
13 Suppl 1:S81-2.
PMID: 10232372
No abstract available.
10.
Gonzalez-Gomez I, Mononen I, Heisterkamp N, Groffen J, Kaartinen V
Am J Pathol
. 1998 Oct;
153(4):1293-300.
PMID: 9777961
Aspartylglycosaminuria (AGU) is one of the most common lysosomal storage disorders in humans. A mouse model for AGU has been recently generated through targeted disruption of the glycosylasparaginase gene, and...