Toshihide Takeuchi
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Explore the profile of Toshihide Takeuchi including associated specialties, affiliations and a list of published articles.
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60
Citations
1849
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Recent Articles
1.
Takakura Y, Hanayama R, Akiyoshi K, Futaki S, Hida K, Ichiki T, et al.
Pharm Res
. 2024 Aug;
41(8):1573-1594.
PMID: 39112776
Extracellular vesicles (EVs) serve as an intrinsic system for delivering functional molecules within our body, playing significant roles in diverse physiological phenomena and diseases. Both native and engineered EVs are...
2.
Ueda T, Takeuchi T, Fujikake N, Suzuki M, Minakawa E, Ueyama M, et al.
Acta Neuropathol Commun
. 2024 Feb;
12(1):20.
PMID: 38311779
The abnormal aggregation of TDP-43 into cytoplasmic inclusions in affected neurons is a major pathological hallmark of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). Although TDP-43 is aberrantly accumulated...
3.
Hirano M, Kuwahara M, Yamagishi Y, Samukawa M, Fujii K, Yamashita S, et al.
Sci Rep
. 2023 Oct;
13(1):17801.
PMID: 37853169
Cerebellar ataxia, neuropathy, and vestibular areflexia syndrome (CANVAS) has recently been attributed to biallelic repeat expansions in RFC1. More recently, the disease entity has expanded to atypical phenotypes, including chronic...
4.
Fujino Y, Ueyama M, Ishiguro T, Ozawa D, Ito H, Sugiki T, et al.
Elife
. 2023 Jul;
12.
PMID: 37461319
Abnormal expansions of GGGGCC repeat sequence in the noncoding region of the gene is the most common cause of familial amyotrophic lateral sclerosis and frontotemporal dementia (C9-ALS/FTD). The expanded repeat...
5.
Choong C, Aguirre C, Kakuda K, Beck G, Nakanishi H, Kimura Y, et al.
Acta Neuropathol
. 2023 Mar;
145(5):573-595.
PMID: 36939875
Lipid interaction with α-synuclein (αSyn) has been long implicated in the pathogenesis of Parkinson's disease (PD). However, it has not been fully determined which lipids are involved in the initiation...
6.
Takeuchi T, Maeta K, Ding X, Oe Y, Takeda A, Inoue M, et al.
Mol Ther Nucleic Acids
. 2023 Feb;
31:353-366.
PMID: 36817728
The abnormal aggregation of TDP-43 into cytoplasmic inclusions in affected neurons is a pathological hallmark of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). Although how TDP-43 forms cytoplasmic aggregates...
7.
Taminato T, Takeuchi T, Ueyama M, Mori K, Ikeda M, Mochizuki H, et al.
Hum Mol Genet
. 2023 Jan;
32(10):1673-1682.
PMID: 36611007
The abnormal expansion of GGGGCC hexanucleotide repeats within the C9orf72 gene is the most common genetic cause of amyotrophic lateral sclerosis (ALS) and frontotemporal dementia (FTD). The accumulation of GGGGCC...
8.
Zhang X, Takeuchi T, Takeda A, Mochizuki H, Nagai Y
PLoS One
. 2022 Jun;
17(6):e0270634.
PMID: 35749554
Extracellular vesicles (EVs) have attracted much attention as potential diagnostic biomarkers for human diseases. Although both plasma and serum are utilized as a source of blood EVs, it remains unclear...
9.
Hatanaka Y, Umeda T, Shigemori K, Takeuchi T, Nagai Y, Tomiyama T
Biomedicines
. 2022 May;
10(5).
PMID: 35625816
The non-coding GGGGCC hexanucleotide repeat expansion (HRE) in gene is a dominant cause of frontotemporal dementia (FTD) and amyotrophic lateral sclerosis (ALS). This intronic mutation elicits the formation of nuclear...
10.
Takeuchi T, Nagai Y
Neurochem Int
. 2022 May;
157:105357.
PMID: 35525394
Polyglutamine (PolyQ) diseases are a group of inherited neurodegenerative diseases including Huntington's disease and several types of spinocerebellar ataxias, which are caused by aggregation and accumulation of the disease-causative proteins...