Thomas M Vondriska
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Explore the profile of Thomas M Vondriska including associated specialties, affiliations and a list of published articles.
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99
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2718
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Recent Articles
1.
Li Z, LaPenna K, Gehred N, Yu X, Tang W, Doiron J, et al.
bioRxiv
. 2025 Jan;
PMID: 39763741
Background: Recent reports suggest increased myocardial iNOS expression leads to excessive protein -nitrosylation, contributing to the pathophysiology of HFpEF. However, the relationship between NO bioavailability, dynamic regulation of protein -nitrosylation...
2.
Kimball T, Gromova T, Gehred N, Chapski D, Wang K, Vaseghi M, et al.
bioRxiv
. 2024 Oct;
PMID: 39464022
Catecholamine dysregulation is a common feature of multiple acute and chronic cardiac conditions, including heart failure. To investigate the role of altered α-adrenergic stimulation on cardiac function, we developed a...
3.
"Circulating Inflammation" and the Plasma Proteome in Heart Failure With Preserved Ejection Fraction
Vondriska T, Lefer D
JACC Basic Transl Sci
. 2024 Oct;
9(9):1085-1087.
PMID: 39444925
No abstract available.
4.
Doiron J, Elbatreek M, Xia H, Yu X, Gehred N, Gromova T, et al.
bioRxiv
. 2024 Sep;
PMID: 39345440
Background: Heart failure with preserved ejection fraction (HFpEF) is a significant public health concern with limited treatment options. Dysregulated nitric oxide-mediated signaling has been implicated in HFpEF pathophysiology, however, little...
5.
Lazaropoulos M, Gibb A, Chapski D, Nair A, Reiter A, Roy R, et al.
Nat Cardiovasc Res
. 2024 Aug;
3(7):869-882.
PMID: 39196175
Differentiation of cardiac fibroblasts to myofibroblasts is necessary for matrix remodeling and fibrosis in heart failure. We previously reported that mitochondrial calcium signaling drives α-ketoglutarate-dependent histone demethylation, promoting myofibroblast formation....
6.
Gural B, Kirkland L, Hockett A, Sandroni P, Zhang J, Rosa-Garrido M, et al.
bioRxiv
. 2024 Aug;
PMID: 39149394
Background: Recent advances in single cell sequencing have led to an increased focus on the role of cell-type composition in phenotypic presentation and disease progression. Cell-type composition research in the...
7.
Fischer M, Arrieta A, Angelini M, Soehalim E, Chapski D, Shemin R, et al.
J Am Heart Assoc
. 2024 Jun;
13(12):e034896.
PMID: 38860408
No abstract available.
8.
Arrieta A, Chapski D, Reese A, Kimball T, Song K, Rosa-Garrido M, et al.
J Biol Chem
. 2024 Jun;
300(7):107434.
PMID: 38830405
During postnatal cardiac hypertrophy, cardiomyocytes undergo mitotic exit, relying on DNA replication-independent mechanisms of histone turnover to maintain chromatin organization and gene transcription. In other tissues, circadian oscillations in nucleosome...
9.
Hu S, Chapski D, Gehred N, Kimball T, Gromova T, Flores A, et al.
Nat Cardiovasc Res
. 2024 May;
3(4):441-459.
PMID: 38765203
Tuning of genome structure and function is accomplished by chromatin-binding proteins, which determine the transcriptome and phenotype of the cell. Here we investigate how communication between extracellular stress and chromatin...
10.
Fischer M, Arrieta A, Angelini M, Soehalim E, Chapski D, Shemin R, et al.
bioRxiv
. 2024 Feb;
PMID: 38352455
Postoperative atrial fibrillation (POAF) is the most common complication after cardiac surgery and a significant cause of increased morbidity and mortality. The development of novel POAF therapeutics has been limited...