T Tennenbaum
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Explore the profile of T Tennenbaum including associated specialties, affiliations and a list of published articles.
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34
Citations
1131
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Recent Articles
1.
Breitkreutz D, Braiman-Wiksman L, Daum N, Denning M, Tennenbaum T
J Cancer Res Clin Oncol
. 2007 Jul;
133(11):793-808.
PMID: 17661083
The protein kinase C (PKC) family represents a large group of phospholipid dependent enzymes catalyzing the covalent transfer of phosphate from ATP to serine and threonine residues of proteins. Phosphorylation...
2.
Braiman L, Alt A, Kuroki T, Ohba M, Bak A, Tennenbaum T, et al.
Mol Cell Biol
. 2001 Oct;
21(22):7852-61.
PMID: 11604519
Insulin stimulates glucose uptake into skeletal muscle tissue mainly through the translocation of glucose transporter 4 (GLUT4) to the plasma membrane. The precise mechanism involved in this process is presently...
3.
Spravchikov N, Sizyakov G, Gartsbein M, Accili D, Tennenbaum T, WERTHEIMER E
Diabetes
. 2001 Jun;
50(7):1627-35.
PMID: 11423485
Altered skin wound healing is a common cause of morbidity and mortality among diabetic patients. However, the molecular mechanisms whereby diabetes alters skin physiology have not been elucidated. In this...
4.
Alt A, Ohba M, Li L, Gartsbein M, Belanger A, Denning M, et al.
Cancer Res
. 2001 Jun;
61(11):4591-8.
PMID: 11389095
In mammalian epidermis, expression of the alpha6beta4 integrin is restricted to the hemidesmosome complexes, which connect the proliferative basal cell layer with the underlying basement membrane. Keratinocyte differentiation is associated...
5.
Liu Y, Paz K, Herschkovitz A, Alt A, Tennenbaum T, Sampson S, et al.
J Biol Chem
. 2001 Mar;
276(17):14459-65.
PMID: 11278339
Incubation of rat hepatoma Fao cells with insulin leads to a transient rise in Tyr phosphorylation of insulin receptor substrate (IRS) proteins. This is followed by elevation in their P-Ser/Thr...
6.
Shen S, Alt A, WERTHEIMER E, Gartsbein M, Kuroki T, Ohba M, et al.
Diabetes
. 2001 Mar;
50(2):255-64.
PMID: 11272134
Insulin and insulin-like growth factor-1 (IGF-1) are members of the family of the insulin family of growth factors, which activate similar cellular downstream pathways. In this study, we analyzed the...
7.
Braiman L, Alt A, Kuroki T, Ohba M, Bak A, Tennenbaum T, et al.
Mol Endocrinol
. 2001 Mar;
15(4):565-74.
PMID: 11266508
Certain protein kinase C (PKC) isoforms, in particular PKCs beta II, delta, and zeta, are activated by insulin stimulation. In primary cultures of skeletal muscle, PKCs beta II and zeta,...
8.
WERTHEIMER E, Spravchikov N, Trebicz M, Gartsbein M, Accili D, Avinoah I, et al.
Endocrinology
. 2001 Feb;
142(3):1234-41.
PMID: 11181540
Impaired wound healing of skin is one of the most serious complications of diabetes. However, the pathogenesis of this process is not known, and it is unclear whether impaired insulin...
9.
Shen S, WERTHEIMER E, Sampson S, Tennenbaum T
J Invest Dermatol
. 2000 Dec;
115(6):949-54.
PMID: 11121124
Skin is one of the major tissues displaying chronic diabetic complications. We have studied glucose transport following stimulation with insulin and IGF-1 in cultured mouse keratinocytes. In proliferating cells, acute...
10.
WERTHEIMER E, Trebicz M, Eldar T, Gartsbein M, Tennenbaum T
J Invest Dermatol
. 2000 Jul;
115(1):24-9.
PMID: 10886503
The insulin receptor and the insulin-like growth factor-1 receptor are widely expressed tyrosine kinases that mediate insulin and insulin-like growth factor-1 signaling. Both receptors are expressed in many cells in...