Susan Schiavi
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Explore the profile of Susan Schiavi including associated specialties, affiliations and a list of published articles.
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14
Citations
1613
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Recent Articles
1.
Graciolli F, Neves K, Barreto F, Barreto D, Dos Reis L, Canziani M, et al.
Kidney Int
. 2017 Mar;
91(6):1436-1446.
PMID: 28318623
Chronic Kidney Disease (CKD)-Mineral and Bone Disorder (CKD-MBD) is a complex disease that is not completely understood. However, some factors secreted by the osteocytes might play an important role in...
2.
Ferreira J, Ferrari G, Neves K, Cavallari R, Dominguez W, Dos Reis L, et al.
PLoS One
. 2013 Nov;
8(11):e79721.
PMID: 24236156
High phosphate intake is known to aggravate renal osteodystrophy along various pathogenetic pathways. Recent studies have raised the possibility that dysregulation of the osteocyte Wnt/β-catenin signaling pathway is also involved...
3.
Jia T, Olauson H, Lindberg K, Amin R, Edvardsson K, Lindholm B, et al.
BMC Nephrol
. 2013 May;
14:116.
PMID: 23718816
Background: In vivo models of uremia are important tools to study numerous aspects of acute and chronic kidney disease. Mouse models are pivotal because most genetically engineered animal models are...
4.
Vassiliadis J, Bracken C, Matthews D, OBrien S, Schiavi S, Wawersik S
J Am Soc Nephrol
. 2011 Jul;
22(8):1453-61.
PMID: 21784900
Alterations to the structure of the glomerular filtration barrier lead to effacement of podocyte foot processes, leakage of albumin, and the development of proteinuria. To better understand the signaling pathways...
5.
Doi S, Zou Y, Togao O, Pastor J, John G, Wang L, et al.
J Biol Chem
. 2011 Jan;
286(10):8655-8665.
PMID: 21209102
Fibrosis is a pathological process characterized by infiltration and proliferation of mesenchymal cells in interstitial space. A substantial portion of these cells is derived from residing non-epithelial and/or epithelial cells...
6.
Feng J, Ye L, Schiavi S
Curr Opin Nephrol Hypertens
. 2009 May;
18(4):285-91.
PMID: 19448536
Osteocytes, the terminally differentiated cell of the osteoblast lineage, account for over 90% of all bone cells. Due to their relative inaccessibility within mineralized matrix, little is known regarding their...
7.
Moller C, Wei C, Altintas M, Li J, Greka A, Ohse T, et al.
J Am Soc Nephrol
. 2006 Dec;
18(1):29-36.
PMID: 17167110
Injury to podocytes and their slit diaphragms typically leads to marked proteinuria. Mutations in the TRPC6 gene that codes for a slit diaphragm-associated, cation-permeable ion channel have been shown recently...
8.
Kurosu H, Ogawa Y, Miyoshi M, Yamamoto M, Nandi A, Rosenblatt K, et al.
J Biol Chem
. 2006 Jan;
281(10):6120-3.
PMID: 16436388
The aging suppressor gene Klotho encodes a single-pass transmembrane protein. Klotho-deficient mice exhibit a variety of aging-like phenotypes, many of which are similar to those observed in fibroblast growth factor-23...
9.
Berndt T, Schiavi S, Kumar R
Am J Physiol Renal Physiol
. 2005 Nov;
289(6):F1170-82.
PMID: 16275744
Phosphate ions are critical for normal bone mineralization, and phosphate plays a vital role in a number of other biological processes such as signal transduction, nucleotide metabolism, and enzyme regulation....
10.
Berndt T, Bielesz B, Craig T, Tebben P, Bacic D, Wagner C, et al.
Pflugers Arch
. 2005 Sep;
451(4):579-87.
PMID: 16151791
The phosphatonin, secreted frizzled-related protein-4 (sFRP-4), induces phosphaturia and inhibits 25-hydroxyvitamin D 1alpha-hydroxylase activity normally induced in response to hypophosphatemia. To determine the mechanism by which sFRP-4 alters renal phosphate...