Stephanie Z Xie
Overview
Explore the profile of Stephanie Z Xie including associated specialties, affiliations and a list of published articles.
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Articles
22
Citations
699
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Recent Articles
1.
Itkin T, Houghton S, Schreiner R, Lin Y, Badwe C, Voisin V, et al.
Nat Immunol
. 2025 Feb;
26(3):378-390.
PMID: 40000903
Transition between activation and quiescence states in hematopoietic stem and progenitor cells (HSPCs) is tightly governed by cell-intrinsic means and microenvironmental co-adaptation. Although this balance is fundamental for lifelong hematopoiesis...
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Comazzetto S, Cassidy D, DeVilbiss A, Jeffery E, Ottesen B, Reyes A, et al.
Blood
. 2024 Oct;
145(1):114-126.
PMID: 39437548
Ascorbate (vitamin C) limits hematopoietic stem cell (HSC) function and suppresses leukemia development, partly by promoting the function of the Tet2 tumor suppressor. In humans, ascorbate is obtained from the...
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Jakobsen N, Turkalj S, Zeng A, Stoilova B, Metzner M, Rahmig S, et al.
Cell Stem Cell
. 2024 Jun;
31(8):1127-1144.e17.
PMID: 38917807
Clonal hematopoiesis (CH) arises when hematopoietic stem cells (HSCs) acquire mutations, most frequently in the DNMT3A and TET2 genes, conferring a competitive advantage through mechanisms that remain unclear. To gain...
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Hosseini M, Voisin V, Chegini A, Varesi A, Cathelin S, Manikoth Ayyathan D, et al.
Res Sq
. 2024 Feb;
PMID: 38405837
Clonal hematopoiesis (CH) arises when a hematopoietic stem cell (HSC) acquires a mutation that confers a competitive advantage over wild-type (WT) HSCs, resulting in its clonal expansion. Individuals with CH...
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Itkin T, Houghton S, Schreiner R, Lin Y, Badwe C, Voisin V, et al.
bioRxiv
. 2023 Apr;
PMID: 37034724
Transition between activation and quiescence programs in hematopoietic stem and progenitor cells (HSC/HSPCs) is perceived to be governed intrinsically and by microenvironmental co-adaptation. However, HSC programs dictating both transition and...
9.
Lim I, Nagree M, Xie S
Essays Biochem
. 2022 Aug;
66(4):333-344.
PMID: 35996953
Acute myeloid leukemia (AML) is a heterogeneous disease of impaired myeloid differentiation and a caricature of normal hematopoiesis. Leukemic stem cells (LSCs) are responsible for long-term clonal propagation in AML...
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