Shireen Hossain
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Explore the profile of Shireen Hossain including associated specialties, affiliations and a list of published articles.
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12
Citations
247
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Recent Articles
1.
Akerman S, Hossain S, Shobo A, Zhong Y, Jourdain R, Hancock M, et al.
J Alzheimers Dis
. 2019 Apr;
69(2):463-478.
PMID: 31006686
There is increasing evidence suggesting that amyloidogenic proteins might form deposits in non-neuronal tissues in neurodegenerative disorders such as Alzheimer's or Parkinson's diseases. However, the detection of these aggregation-prone proteins...
2.
Maysinger D, Ji J, Moquin A, Hossain S, Hancock M, Zhang I, et al.
ACS Chem Neurosci
. 2017 Oct;
9(2):260-271.
PMID: 29078046
Dendritic polyglycerols (dPG), particularly dendritic polyglycerol sulfates (dPGS), have been intensively studied due to their intrinsic anti-inflammatory activity. As related to brain pathologies involving neuroinflammation, the current study examined if...
3.
Rao V, Khan D, Cui Q, Fuh S, Hossain S, Almazan G, et al.
PLoS One
. 2017 Aug;
12(8):e0182372.
PMID: 28792512
Within the microenvironment of multiple sclerosis lesions, oligodendrocytes are subject to metabolic stress reflecting effects of focal ischemia and inflammation. Previous studies have shown that under optimal conditions in vitro,...
4.
Prade E, Barucker C, Sarkar R, Althoff-Ospelt G, Lopez Del Amo J, Hossain S, et al.
Biochemistry
. 2016 Feb;
55(12):1839-49.
PMID: 26900939
Alzheimer's disease is characterized by deposition of the amyloid β-peptide (Aβ) in brain tissue of affected individuals. In recent years, many potential lead structures have been suggested that can potentially...
5.
Barucker C, Bittner H, Chang P, Cameron S, Hancock M, Liebsch F, et al.
Sci Rep
. 2015 Oct;
5:15410.
PMID: 26510576
The amyloid-β42 (Aβ42) peptide is believed to be the main culprit in the pathogenesis of Alzheimer disease (AD), impairing synaptic function and initiating neuronal degeneration. Soluble Aβ42 oligomers are highly...
6.
Hossain S, Liu H, Fragoso G, Almazan G
Neuropharmacology
. 2014 Jan;
79:506-14.
PMID: 24412648
Prolonged exposure of oligodendrocyte progenitor cultures to non-toxic concentrations of glutamate receptor agonists for 24 h decreased cellular proliferation mediated by α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors. Since prolonged agonist stimulation can...
7.
De Paula M, Cui Q, Hossain S, Antel J, Almazan G
Glia
. 2013 Oct;
62(1):64-77.
PMID: 24166839
Oligodendrocytes (OLGs) produce and maintain myelin in the central nervous system (CNS). In the demyelinating autoimmune disease multiple sclerosis, OLGs are damaged and those remaining fail to fully remyelinate CNS...
8.
Hossain S, de la Cruz-Morcillo M, Sanchez-Prieto R, Almazan G
Glia
. 2012 Apr;
60(7):1130-44.
PMID: 22511272
We previously reported that addition of extracellular matrix (ECM) extracts to rat Schwann cell-dorsal root ganglion neuron (DRGN) co-cultures activated mitogen-activated protein kinase (MAPK) p38, whereas inhibition blocked myelination. Here,...
9.
Hossain S, Fragoso G, Mushynski W, Almazan G
Exp Neurol
. 2010 Aug;
226(1):47-57.
PMID: 20696161
Fyn, a nonreceptor Src-like tyrosine kinase (SLK), plays an important role in oligodendrocyte differentiation and myelination in the brain. However, its role in myelination of peripheral nerves remains undefined. Here...
10.
Hossain S, Liu H, Nguyen M, Shore G, Almazan G
Neurotoxicology
. 2009 Jun;
30(4):544-54.
PMID: 19523979
Cadmium toxicity has been associated with learning disabilities and Parkinsonian symptoms in humans. We have previously shown that cultured oligodendrocytes are directly damaged by cadmium exposure. Here, we characterized the...