Shane R Nelson
Overview
Explore the profile of Shane R Nelson including associated specialties, affiliations and a list of published articles.
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25
Citations
519
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Recent Articles
1.
Mead A, Wood N, Nelson S, Palmer B, Yang L, Previs S, et al.
J Gen Physiol
. 2024 Nov;
156(12).
PMID: 39535407
No abstract available.
2.
Mead A, Wood N, Nelson S, Palmer B, Yang L, Previs S, et al.
J Gen Physiol
. 2024 Oct;
156(12).
PMID: 39373654
Myosin-binding protein H (MyBP-H) is a component of the vertebrate skeletal muscle sarcomere with sequence and domain homology to myosin-binding protein C (MyBP-C). Whereas skeletal muscle isoforms of MyBP-C (fMyBP-C,...
3.
Mead A, Wood N, Nelson S, Palmer B, Yang L, Previs S, et al.
bioRxiv
. 2024 May;
PMID: 38798399
Myosin-binding protein H (MyBP-H) is a component of the vertebrate skeletal muscle sarcomere with sequence and domain homology to myosin-binding protein C (MyBP-C). Whereas skeletal muscle isoforms of MyBP-C (fMyBP-C,...
4.
Tail length and E525K dilated cardiomyopathy mutant alter human β-cardiac myosin super-relaxed state
Duno-Miranda S, Nelson S, Rasicci D, Bodt S, Cirilo Jr J, Vang D, et al.
J Gen Physiol
. 2024 May;
156(6).
PMID: 38709176
Dilated cardiomyopathy (DCM) is a condition characterized by impaired cardiac function, due to myocardial hypo-contractility, and is associated with point mutations in β-cardiac myosin, the molecular motor that powers cardiac...
5.
Tail Length and E525K Dilated Cardiomyopathy Mutant Alter Human β-Cardiac Myosin Super-Relaxed State
Duno-Miranda S, Nelson S, Rasicci D, Bodt S, Cirilo Jr J, Vang D, et al.
bioRxiv
. 2023 Dec;
PMID: 38105932
Dilated cardiomyopathy (DCM) is characterized by impaired cardiac function due to myocardial hypo-contractility and is associated with point mutations in β-cardiac myosin, the molecular motor that powers cardiac contraction. Myocardial...
6.
Stadler R, Nelson S, Warshaw D, Ward G
Elife
. 2022 Dec;
11.
PMID: 36519527
is a protozoan parasite that infects 30-40% of the world's population. Infections are typically subclinical but can be severe and, in some cases, life threatening. Central to the virulence of...
7.
Nelson S, Li A, Beck-Previs S, Kennedy G, Warshaw D
Biophys J
. 2020 Aug;
119(6):1050-1055.
PMID: 32857963
Striated muscle contraction is the result of sarcomeres, the basic contractile unit, shortening because of hydrolysis of adenosine triphosphate (ATP) by myosin molecular motors. In noncontracting, "relaxed" muscle, myosin still...
8.
Lee A, Majumdar C, Kathe S, Van Ostrand R, Vickery H, Averill A, et al.
J Am Chem Soc
. 2020 Jul;
142(31):13283-13287.
PMID: 32664726
MutY glycosylase excises adenines misincorporated opposite the oxidatively damaged lesion, 8-oxo-7,8-dihydroguanine (OG), to initiate base excision repair and prevent G to T transversion mutations. Successful repair requires MutY recognition of...
9.
Li A, Nelson S, Rahmanseresht S, Braet F, Cornachione A, Previs S, et al.
Proc Natl Acad Sci U S A
. 2019 Oct;
116(43):21882-21892.
PMID: 31591218
Skeletal muscle myosin-binding protein C (MyBP-C) is a myosin thick filament-associated protein, localized through its C terminus to distinct regions (C-zones) of the sarcomere. MyBP-C modulates muscle contractility, presumably through...
10.
Lombardo A, Nelson S, Kennedy G, Trybus K, Walcott S, Warshaw D
Proc Natl Acad Sci U S A
. 2019 Apr;
116(17):8326-8335.
PMID: 30967504
The cell's dense 3D actin filament network presents numerous challenges to vesicular transport by teams of myosin Va (MyoVa) molecular motors. These teams must navigate their cargo through diverse actin...