Scott E Nelson
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Explore the profile of Scott E Nelson including associated specialties, affiliations and a list of published articles.
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Articles
19
Citations
235
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Recent Articles
1.
Singh D, Zhang Y, White S, Nelson S, Fogarty S, Pawelski A, et al.
PLoS Pathog
. 2025 Feb;
21(2):e1012934.
PMID: 39951426
EBV infects normal oral keratinocytes (NOKs) and plays an essential role in undifferentiated nasopharyngeal carcinoma (NPC). We previously showed that the EBV oncogene, LMP1, promotes proliferation and inhibits spontaneous differentiation...
2.
Bristol J, Nelson S, Ohashi M, Casco A, Hayes M, Ranheim E, et al.
PLoS Pathog
. 2024 Apr;
20(4):e1012132.
PMID: 38620028
Epstein-Barr virus (EBV) is an important cause of human lymphomas, including Burkitt lymphoma (BL). EBV+ BLs are driven by Myc translocation and have stringent forms of viral latency that do...
3.
Singh D, Nelson S, Pawelski A, Kansra A, Fogarty S, Bristol J, et al.
Proc Natl Acad Sci U S A
. 2023 May;
120(20):e2219755120.
PMID: 37155846
Latent Epstein-Barr virus (EBV) infection promotes undifferentiated nasopharyngeal carcinomas (NPCs) in humans, but the mechanism(s) for this effect has been difficult to study because EBV cannot transform normal epithelial cells...
4.
Singh D, Nelson S, Pawelski A, Cantres-Velez J, Kansra A, Pauly N, et al.
PLoS Pathog
. 2022 Oct;
18(10):e1010868.
PMID: 36190982
Differentiated epithelial cells are an important source of infectious EBV virions in human saliva, and latent Epstein-Barr virus (EBV) infection is strongly associated with the epithelial cell tumor, nasopharyngeal carcinoma...
5.
Beauvais D, Nelson S, Adams K, Stueven N, Jung O, Rapraeger A
J Biol Chem
. 2022 May;
298(6):102029.
PMID: 35569509
Epidermal growth factor receptor (EGFR) is a causal factor in carcinoma, yet many carcinoma patients are resistant to EGFR inhibitors. Potential insight into this resistance stems from prior work that...
6.
Bristol J, Brand J, Ohashi M, Eichelberg M, Casco A, Nelson S, et al.
PLoS Pathog
. 2022 Apr;
18(4):e1010453.
PMID: 35472072
Humans are infected with two types of EBV (Type 1 (T1) and Type 2 (T2)) that differ substantially in their EBNA2 and EBNA 3A/B/C latency proteins and have different phenotypes...
7.
Van Sciver N, Ohashi M, Pauly N, Bristol J, Nelson S, Johannsen E, et al.
PLoS Pathog
. 2021 Aug;
17(8):e1009783.
PMID: 34339458
The Epstein-Barr virus (EBV) human herpesvirus is associated with B-cell and epithelial-cell malignancies, and both the latent and lytic forms of viral infection contribute to the development of EBV-associated tumors....
8.
Li C, Romero-Masters J, Huebner S, Ohashi M, Hayes M, Bristol J, et al.
PLoS Pathog
. 2020 Jun;
16(6):e1008590.
PMID: 32542010
EBV transforms B cells in vitro and causes human B-cell lymphomas including classical Hodgkin lymphoma (CHL), Burkitt lymphoma (BL) and diffuse large B-cell lymphoma (DLBCL). The EBV latency protein, EBNA2,...
9.
Romero-Masters J, Ohashi M, Djavadian R, Eichelberg M, Hayes M, Zumwalde N, et al.
J Virol
. 2020 Mar;
94(10).
PMID: 32132242
Epstein-Barr virus (EBV) causes B cell lymphomas and transforms B cells The EBV protein EBNA3A collaborates with EBNA3C to repress p16 expression and is required for efficient transformation An EBNA3A...
10.
Romero-Masters J, Huebner S, Ohashi M, Bristol J, Benner B, Barlow E, et al.
PLoS Pathog
. 2020 Feb;
16(2):e1008365.
PMID: 32059024
Humans are infected with two distinct strains (Type 1 (T1) and Type 2 (T2)) of Epstein-Barr virus (EBV) that differ substantially in their EBNA2 and EBNA 3A/B/C latency genes and...